Dr. Gordon’s Comments:

A contributor to food allergies/sensitivities has emerged, tick bites. This is a great piece of detective work and seems to not affect B or AB blood groups, as much as Type A or O.

“Scott Commins, an assistant professor of medicine and lead author of the U-Va. Study published in the Journal of Allergy and Clinical Immunology, said that in susceptible people such as Newell, a tick bite that causes a significant skin reaction seems to trigger the production of an antibody that binds to a sugar present on meat called alpha-galactosidase, also known as alpha-gal. When a person who has the antibody eats meat, it triggers the release of histamine, which causes the allergic symptoms: hives, itching and, in the worst case,alpha-galactosidase.”

Of course today with our epidemic of autoimmune disease and leaky gut and low level infections like Chlamydia and CMV in almost everyone, these elevated antibodies to something or even auto-antibodies are very common and may be more dangerous than widely appreciated.

I still encourage those of you who are sensitive to so many foods to understand that leaky gut is epidemic today. I am convinced GMO foods are a contributor, as they introduce Bacillus Theringensis into our bodies, which are now detectible in the blood stream of patients! Bt then causes Dysbiosis.

I hope many of you will try my concept of using my Detox Drink (BioEn’R-G’y C, H Minus, ZeoGold) with my Power Drink and acidophilus to have a healthy gut and be better able to handle the toxic mess our Standard American Diet has become. Remember high levels or the wrong fats from corn and soy etc. set the stage for chronic inflammation. I believe that if this patient wanted to one day eat beef or fish without his allergic reaction without any doubt my F.I.G.H.T.E.M with M.I.C.E. program would get him healthy again and he would not have to suffer these life threatening allergic reactions.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com

Link: http://www.washingtonpost.com/wp-dyn/content/article/2009/10/19/AR2009101902874.html

Excerpt:

MEDICAL MYSTERIES
Man’s Sudden Food Allergy Was a Medical Mystery for Months
By Sandra G. Boodman Special to The Washington Post
Tuesday, October 20, 2009

This cannot be happening again, Hayden Newell thought as the angry, red, ferociously itchy welts encircled his waist and spread up his arms. The 57-year-old metallurgist from tiny Boones Mill, Va., who was attending a business lunch in Florida, knew what would probably happen next: His lips would grow numb, making it hard to speak, he would become short of breath and his blood pressure would plummet: all unmistakable signs of anaphylaxis, a potentially fatal allergic reaction. Newell knew from experience that he had to get to an emergency room — fast.

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http://www.resultsrna.com/products/acs_200_proven_to_kill_borrelia_burgdorferi.php

The bacterial organism behind a newly identified tick-borne illness found halfway across the world showed up in Durland Fish’s lab in Connecticut a decade ago.

http://www.ctmirror.org/story/13955/lyme-new-tick-borne-disease-yale

Strange????????? Fishboy, should be locked up to protect us….FOLLOW THE MONEY!!

ABSTRACT: BACKGROUND: There are few published reports on canine Babesia,
Ehrlichia, Anaplasma, Hepatozoon and haemotropic Mycoplasma infections
in India and most describe clinical disease in individual dogs,
diagnosed by morphological observation of the microorganisms in stained
blood smears. This study investigated the occurrence and distribution of
canine tick-borne disease (TBD) pathogens using a combination of
conventional and molecular diagnostic techniques in four cities in India.

Continuous exposure to intrathecal B. burgdorferi OspC
led to activation of microglia and axonal damage without demonstrable
cognitive impairment in experimental mice. These results suggest that
long-term intrathecal exposure to OspC resulted in axonal damage that
may underlie the neurologic manifestations in chronic LNB.

 This paralleled greater Borreli a burden in
γδ-deficient mice as well as more cardiac inflammation. These findings
are consistent with a model of γδ T cells functioning to promote the
adaptive immune response during infection.

CONCLUSION: Antibiotics have varying effects on the different
morphological forms of B. burgdorferi. Persistence of viable organisms
in round body forms and biofilm-like colonies may explain treatment
failure and persistent symptoms following antibiotic therapy of Lyme
disease.

Excerpt:

CONCLUSIONS: We found that the time of the year, associated neurological
symptoms and mononuclear pleocytosis were strong predictive factors for
Lyme neuroborreliosis as a cause of peripheral facial palsy in an area
endemic for Borrelia.