Linda’s comment:   We all hear about GMO foods and how deadly it can be for those of us with chronic illness….We have an epidemic of poor health, yet we can fix that if we clean up our diets….

Dr. Gordon’s Comments:

GMO foods are one cause of todays epidemic of poor health contributing to food sensitivities is the tip of the iceberg. Now, there is hope we can stop this monster; truth in labeling will put an end to this travesty.

“Why are there basically no genetically engineered foods or crops anywhere in Europe, while 75 percent of U.S. supermarket foods—including many so-called “natural” foods—are GE tainted?

The answer is simple. In Europe genetically modified foods and ingredients have to be labeled. In the U.S. they do not. Up until now, in North America, Monsanto and the Biotechnocrats have enjoyed free reign to secretly lace non-organic foods with gene-spliced viruses, bacteria, antibiotic-resistant marker genes, and foreign DNA—mutant “Frankenfoods” shown to severely damage the health of animals, plants, and other living organisms in numerous scientific studies.

Monsanto and their allies understand the threat that truth-in-labeling poses for GMOs”

See the attached and know that together consumers do have the power to end Monsanto control of all food.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com

Link: http://articles.mercola.com/sites/articles/archive/2011/10/03/cbi-taking-down-monsanto-gmo-products.aspx?e_cid=20111003_DNL_art_1

Excerpt:

By: Ronnie Cummins
Organic Consumers Association

“If you put a label on genetically engineered food you might as well put a skull and crossbones on it.” – Norman Braksick, president of Asgrow Seed Co., a subsidiary of Monsanto, quoted in the Kansas City Star, March 7, 1994

Monsanto and Food Inc.’s stranglehold over the nation’s food and farming system is about to be challenged in a food fight that will largely determine the future of American agriculture.

From: Jeffrey Smith

Celebrating the Non-GMO Revolution

Dear Garry,

On January 20th Pamm Larry had an epiphany, as she calls it. Why not have a GMO labeling law on the November 2012 ballot in California? She did her research, put a website together, and “came out” on March 20—as a single voice with a big idea. Within six months, there were about 70 state leaders – holding events, showing films, recruiting support, and creating the framework for what may become the pivotal vote to usher GMOs out of our food supply.

Linda’s comment:  It is nice to see more Vets are getting involved with Lyme and pets….

Excerpt:

The present study describes the only deer
piroplasm detected so far that shows complete identity with B. divergens, in
just over half of the 18S rRNA gene. The entire gene of this deer parasite
should be analysed and transmission experiments undertaken before the
infectivity of B. divergens for red deer can be confirmed.

 

Link: http://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=21314977&retmode=ref&cmd=prlinks

Linda’s comments: Linda’s comment:  This is the reasons why I try and guide people to make their own healthcare choices and learn all they can about holistic/alternative medicine….There are wayyyyyyyyyy to many folks with Lyme disease who are given these products…..<sigh>

link: http://www.medicationsense.com/fluoroquinolone.html 

Excerpt:

Levaquin and Cipro Reactions

In 2001, Dr. Jay S. Cohen published a ground-breaking article* on the severe and often disabling reactions some people sustained while taking Levaquin, Cipro, or another FQ antibiotic. Dr. Cohen says, “It is difficult to describe the severity of these reactions. They are devastating. Many of the people in my study were healthy before their reactions. Some were high intensity athletes. Suddenly they were disabled, in terrible pain, unable to work, walk, or sleep.” 
The 45 subjects in Dr. Cohen’s study reported the following side effects*.

Peripheral Nervous System

: Tingling, numbness, prickling, burning pain, pins/needles sensation, electrical or shooting pain, skin crawling, sensation, hyperesthesia, hypoesthesia, allodynia (sensitivity to touch), numbness, weakness, twitching, tremors, spasms.

Central Nervous System:

 

Dizziness, malaise, weakness, impaired coordination, nightmares, insomnia, headaches, agitation, anxiety, panic attacks, disorientation, impaired concentration or memory, confusion, depersonalization, hallucinations, psychoses.

Navy researcher links toxins in war-zone dust to ailments

By Kelly Kennedy, USA TODAY

U.S. troops in Iraq, Afghanistan and Kuwait have inhaled microscopic dust particles laden with toxic metals, bacteria and fungi — a toxic stew that may explain everything from the undiagnosed Gulf War Syndrome symptoms lingering from the 1991 war against Iraq to high rates of respiratory, neurological and heart ailments encountered in the current wars, scientists say.

“From my research and that of others, I really think this may be the smoking gun,” says Navy Capt. Mark Lyles, chair of medical sciences and biotechnology at the Center for Naval Warfare Studies at the Naval War College in Newport, R.I. “It fits everything — symptoms, timing, everything.”

Excerpt:

Nature.com

It’s time for animals – including humans – to admit that the bacteria, viruses and other microbes have won. Our bodies are home to many times more bacterial cells than animal cells and countless trillions of viruses. Ancient retroviruses make up a good size chunk of our genome. Now, scientists have discovered that most any virus can set up shop in an animal’s genomes and lay dormant for millions of years.

A scan of 44 mammal genomes, plus those of several mosquito and tick vectors and two birds that could serve as reservoirs, has uncovered DNA sequences that can be traced to 10 different families of viruses, including some related to viruses that cause hepatitis B, Ebola, rabies and dengue. Most of the viral sequences are riddled with enough mutations to be considered junk, but some appear to encoding working genes co-opted by their host. The work is published online today in the journal PLoS Genetics.

It’s not obvious how all these viruses got into animal genomes. The researchers, Aris Katzourakis at the University of Oxford, UK, and Robert Gifford at Rockefeller University in New York, searched specifically for viruses that aren’t retroviruses, which are obligated to copy their DNA into hosts. Many but not all of the viruses infect their hosts persistently or replicate inside of the nucleus, however, offering ample opportunity to take up residence in the genomes of germ cells.

The work is just a first look at all the non-retroviruses in the animal genome, but Katzourakis and Gifford turned up a few interesting findings. For instance, their scan identified sequences from filoviruses, the family Ebola belongs to, in the genomes of bats, tarsiers, several rodents, opossums and even wallabies. This hints that filoviruses have a much wider host range than the primate and bat species which these viruses are known to infect.

The paper also hints at unknown ancient transmissions of viruses between hosts. The bottlenose dolphin genome, it turns out, is home to sequences of a kind of parvovirus similar to one found in birds, suggesting that the viruses may have jumped between mammals and birds in the past.

Most of these sequences are junk, so filled with mutations that they can’t make working proteins. But some of the viral sequences might do something inside their hosts. One example is a bornavirus gene called EBLN-1 that took up residence in ancient primate genomes some 50 million years ago and survives intact in many modern primates, including humans. A similar protein latches onto RNA in bornaviruses, so it might do the same in primates as part of a viral defence mechanism, Gifford speculates.

Like the ancient retroviruses locked inside animal genomes, these viruses offer a window into infections that occurred millions of years ago.

“People who are looking at the ecology of those diseases, they very much work in recent time and they have no assumptions that it’s an old system that might have evolved over billions of years,” says Gifford. “The data that we’re finding is really contradicting that and providing the first evidence that these are really old relationships between hosts and viruses, and I think it’s really critical to how we underestand them to get that context right.”

 

Comments

This is a key paper with major implications. Hundreds of viruses appear to have infiltrated the human genome. The important consequence of this is that the proteins
of today’s viruses resemble our own. Numerous BLASTs of translated viral DNA vs.the human proteome are shown at this site named Pandora’s
box for obvious reasons. Small contigous amino acid stretches of 5 or more amino acids within human proteins exactly match those in the current virome (Vatches = viral matches).
Upon infection these viral proteins are likely to seed havoc within the host’s protein networks, acting as dummy ligands, decoy receptors or by interactome interference. This has major implications for understanding how viruses contribute to disease and several examples are shown.For example it
would appear that such viral insertions,repeated over evolutionary time, are
responsible for the creation of gene families. HSV-1 and HSV-2 are homologous to many kinases,
and the cytomegalovirus to many chemokine receptors.

The viruses implicated as risk factors in Alzheimer’s disease, schizophrenia and Parkinson’s disease all express proteins that are homologous to hundreds
of susceptibity gene products in these diseases.This suggests that genes and risk factors act together, and that each may be a risk factor precisely because of such matches. In addition, given such homology at both the DNA and protein level, it is likely that some gene association studies, using blood samples, have been
indexing infection as well as identifying key susceptibility genes.

Full story: http://news.vin.com/VINNews.aspx?articleId=16570

Excerpt:

The pathogen best known for causing cat scratch disease is responsible for a host of serious illnesses in humans that may be misdiagnosed due to lack of awareness in the medical community.

Researchers studying the Bartonella genus of bacteria say veterinarians and veterinary staff, along with others who work with animals — including groomers, trainers and shelter and rescue organization personnel — are at particular risk of infection owing to their frequent exposure to animals and animal parasites such as fleas.

“I think it’s more common than we think in the veterinary community,” said Dr. Bruno Chomel, a DVM and professor in the Center for Vectorborne Diseases at the University of California, Davis.

The scope and significance of Bartonella infection among humans in veterinary circles and at large is just beginning to be understood.

Full article: http://www.medicalnewstoday.com/articles/196744.php

Excerpt:

A possible cause of irritable bowel syndrome has been traced to a small piece of RNA that blocks a substance protecting the colon membrane, leading to hostile conditions that can produce diarrhea, bloating and chronic abdominal pain.

New research shows that this RNA segment sends signals that stop the activity of the gene that produces glutamine, an amino acid. Previous research has linked a shortage of glutamine in the gut with the seepage of toxins and bacteria through the intestinal wall, irritating nerves and creating disease symptoms.

Scientists say that trying to generate glutamine in the disordered bowel by silencing this RNA segment could open up a whole new way of thinking about treating the diarrhea-predominant type of irritable bowel syndrome (IBS). In the meantime, they are making plans to conduct a clinical trial to see if glutamine supplements could also reduce common IBS symptoms

This form of the disorder is characterized by diarrhea and bloating as well as chronic abdominal pain that is difficult to treat. About a third of IBS patients have the diarrhea-predominant type, another third experience consistent constipation, and the rest experience alternating bouts of diarrhea and constipation.

Full article: http://www.smh.com.au/national/your-body-bugs-are-unique-20100721-10la6.html

Excerpt:

MANY things make us stand out as individuals, from our fingerprints to our iris patterns. Now scientists have discovered another – the viruses in our gut.

People who are closely related tend to have similar bacteria in their intestines. But a study of the viruses in people’s faeces has found that even identical twins have uniquely different populations of these tiny tenants.

Florent Angly, a biologist at the University of Queensland and a co-author of the study, said the findings showed that factors such as health and diet were the biggest influence on the kinds of viruses people harboured.

”Twins have an identical genome, so including twins in the study was designed to find out what is more important: nature or nurture,” he said.

Linda’s comments: Amazing how animals get better research and treatments than humans!!??  God Bless Dr Breitschwerft for his research …… He found “first time documented evidence that the pathogen may have been passed between family members.”  What Lida Mattman said all alone.  Then they say ,”At least 26 strains of Bartonella have been named worldwide, and the list is growing.”…..  AGAIN, a VET finds out with his continued research how deadly Bartonella can be.  This statement/quote ” Dr. Michael Kosoy, who heads the Bartonella laboratory for the Centers for Disease Control and Prevention in Fort Collins, Colo., said scientists are only beginning to build evidence that Bartonella infections may be more common than previously thought.” WHAT, NOW the CDC is admitting that Bartonella exists??  WOW….unbelievable….Perhaps all the pressure the Lymies are putting on research, IDSA and doctors is working….

BY SARAH AVERY – Staff Writer

A bacterial infection typically spread by fleas, lice and biting flies could be more prevalent than many think, and may have been transmitted from a mother to her children at birth, scientists from N.C. State University say.

Dr. Edward Breitschwerdt, an infectious disease veterinarian and one of the world’s leading researchers of bacteria called Bartonella, has for the first time documented evidence that the pathogen may have been passed between family members.

Although more studies are needed to back up his findings, Breitschwerdt and colleagues describe the case of a mother and father who began battling chronic aches, fatigues and other symptoms soon after they were married. When their twins were born in 1998, the daughter died after nine days from a heart defect, and the son developed chronic health problems.

Using tissue from the daughter’s autopsy and blood from the surviving family members, Breitschwerdt’s team discovered that the entire family was infected with the same species of Bartonella bacteria, despite having no shared exposures to flea or lice infestations. Bartonella is known to causes such illnesses as trench fever and cat scratch disease, and it is increasingly suspected of triggering a variety of aches and inflammations that doctors have been unable to diagnose.

“I think we have stumbled across something that is of monumental medical importance,” said Breitschwerdt, whose findings were published recently in the Journal of Clinical Microbiology.

Proving the mother-child transmission could be difficult, however. Little funding is available for such research because the bacteria are still not considered a major source of human disease.

Dr. Michael Kosoy, who heads the Bartonella laboratory for the Centers for Disease Control and Prevention in Fort Collins, Colo., said scientists are only beginning to build evidence that Bartonella infections may be more common than previously thought.

Full article: http://www.plospathogens.org/article/info:doi/10.1371/journal.ppat.1000886

Excerpt:

Humans are colonized by diverse populations of bacteria and fungi when in a healthy state and in the settings of disease, and the interactions between these microbial populations can be beneficial or detrimental to the host [1]. Among these microbial populations, Candida albicans is the fungus most commonly detected in association with humans [2], and numerous studies have described C. albicans interactions with its bacterial neighbors [1]. Here, with a focus on C. albicans, we provide examples of how bacterial-fungal interactions can influence human health. In addition, we highlight studies that give insight into the molecular mechanisms that govern the physical associations, interspecies communication, and changes in microbial behavior and survival that occur when bacteria and fungi occupy the same sites.