bone lead – F.I.G.H.T for your health! http://lymebook.com/fight Linda Heming describes her Lyme disease healing journey Wed, 06 Nov 2013 05:54:37 +0000 en-US hourly 1 https://wordpress.org/?v=4.9.25 Lead in Bone and death http://lymebook.com/fight/lead-in-bone-and-death/ http://lymebook.com/fight/lead-in-bone-and-death/#respond Fri, 26 Mar 2010 06:09:24 +0000 http://lymebook.com/fight/?p=952 Is lead more important that cholesterol levels? This research shows that higher bone lead leads to a six-fold increase in heart attacks.

Bone lead levels reflect LIFE TIME exposures and are clearly the best test, more accurate than blood, urine, hair, provoked levels etc. Adult bones take 15 years average to REMODEL, and we are born with 1000 times greater bone lead levels than existed a few hundred years ago. You will understand why I advocate daily lead removal for 15 years.

Chelation only removes lead from readily mobilized organs and soft tissues stores, not bone. This makes everyone function better, but when you stop, a new equilibrium is achieved and bone lead will download lead again. This means that the benefits from any form of chelation will be short lived.

You must have a long-term program such as Beyond Chelation-Improved, Zeolite or high-dose Vitamin C or Fiber that people will follow consistently for many years if you want to significantly lower the incidence of morbidity and mortality in any population. Lead has been shown to contribute to all causes of morbidity and mortality and your Mom always told you to “GET THE LEAD OUT” anyway.

We are all taking in more lead daily from our water, food and air, and thus we need a program that permits us to eliminate more lead and other heavy metals than we take in. That way, over time, we can all lower our total bone burden and keep it low, including bone lead levels.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com

Full article: http://circ.ahajournals.org/cgi/content/short/120/12/1056

Excerpt:

From the Departments of Environmental Health (M.G.W., J.S.) and Epidemiology (M.G.W., J.S.), Harvard School of Public Health, Boston, Mass; Channing Laboratory, Department of Medicine, Brigham and Women’s Hospital, Harvard Medical School, Boston, Mass (M.G.W., J.S.); Department of Physical Medicine and Rehabilitation, Spaulding Rehabilitation Hospital and Harvard Medical School, Boston, Mass (N.J.); School of Health Sciences, Purdue University, West Lafayette, Ind (H.N.); Department of Veterans Affairs Normative Aging Study, Veterans Affairs Boston Healthcare System, Boston, Mass (D.S., P.V.); Department of Medicine, Boston University School of Medicine, Boston, Mass (D.S., P.V.); Department of Epidemiology, Boston University School of Public Health, Boston, Mass (D.S., P.V.): and Department of Environmental Health Sciences, University of Michigan, Ann Arbor (H.H.).
Received October 8, 2008; accepted July 31, 2009.

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Environmental Lead Exposure http://lymebook.com/fight/environmental-lead-exposure/ Fri, 22 Jan 2010 07:16:28 +0000 http://lymebook.com/fight/environmental-lead-exposure/ Linda’s comment:  Dr Gordon’s FIGHT program will deal with helping to reduce and/or removing lead from the body that will help protect the kidneys.  I have personally been on the FIGHT program for 1 1/2 years and it is the best thing I have ever done….Removing heavy metals from our bodies will help us to protect our immune from disease.  I have posted on Mercury and all the things that it effects….this is serious, please read that post closely. 
 
Regards,
Linda or Angel

 

Many may remember the NEJM article proving that patients with early renal impairment could often avoid renal dialysis if they routinely were maintained on CALCIUM EDTA infusions. Now there is new research finding that low levels of lead from the environment are adversely affecting renal function. By now, hopefully, all of you are aware that Lead levels have been tied to all causes of morbidity and mortality. However, remember the SYNERGISTIC toxicity effects where small amounts of other toxic metals like Mercury dramatically enhance the adverse effect of Lead.

Unfortunately NO test seems to be as accurate as BONE LEAD in determining risks of cataracts or heart attacks. But since we know we are all toxic today, this is just another indicator that may lead a few more patients to take preventive steps with a program using oral or parenteral chelators and or Zeolite, or even high dose oral vitamin c.  Renal dialysis is not fun and kidney transplants are not easy to come by.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com

http://www.medscape.com/viewarticle/715046

From Medscape Medical News

Low-Level Environmental Lead Exposure May Adversely Affect Renal Function
Laurie Barclay, MD

January 13, 2010 — Low-level environmental lead exposure may adversely affect renal function, according to results from the Third National Health and Nutrition Examination Survey, reported in the January 11 issue of the Archives of Internal Medicine.

“Chronic, high-level lead exposure is a known risk factor for kidney disease,” write Jeffrey J. Fadrowski, MD, MHS, from Bloomberg School of Public Health, Johns Hopkins University, Baltimore, Maryland, and colleagues. “The effect of current low-level environmental lead exposure is less well known, particularly among children, a population generally free from kidney disease risk factors such as hypertension and diabetes mellitus. Therefore, in this study, we investigated the association between lead exposure and kidney function in a representative sample of US adolescents.”

The study cohort consisted of 769 adolescents aged 12 to 20 years for whom whole blood lead and serum cystatin C were measured in the Third National Health and Nutrition Examination Survey, which took place from 1988 to 1994. The investigators evaluated the association between blood lead level and level of kidney function, reflected in glomerular filtration rate (GFR) calculated from cystatin C–based and creatinine-based estimating equations.

Median whole blood lead level was 1.5 μg/dL (to convert to micromoles per liter, multiply by 0.0483), and median cystatin C–estimated GFR was 112.9 mL/min/1.73 m2. Compared with participants in the first quartile of lead levels (<1 μg/dL), those with lead levels in the highest quartile (≥3.0 μg/dL) had a lower estimated GFR by 6.6 mL/min/1.73 m2 (95% confidence interval [CI], −0.7 to −12.6 mL/min/1.73 m2). Twofold increase in blood lead level was associated with lower estimated GFR by 2.9 mL/min/1.73 m2 (95% CI, −0.7 to −5.0 mL/min/1.73 m2). Although lead levels were also associated with lower creatinine-based estimated GFR levels, t

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