Lyme is all around us but I believe we will help many more if we give up on blaming everything on the tick related introduction of more pathogens than we had the day before we are bit.  It is confusing to people, as too often Lyme tests are inconclusive. So let’s rename the condition LIMES (Lowered Immune Metabolic Encephalopathy Syndrome) or LIMNS (for Neuropathy, as in MS like conditions) or LIMAS (Arthropathy when it is more arthritic in presentation), as these names move us closer to seeing the true picture.

It is sad to turn patients away with these devastating symptoms when the Infectious Disease Association guidelines force us to say it is Lyme. I am certain broadening the approach to Food sensitivities, other Infections, Genetics, Heavy metals and Hormones and Toxins would wind up with better results than the low batting average that is reported from long-term IV antibiotics, which are often reported as low as 33% about which Lyme critics point out is the response rate to placebos. If we focus on my F.I.G.H.T. program and do something to help deal with the obvious issues that can be found in almost anyone in any of these categories, we can be more cost effective and actually help more patients, as they will stop looking just for a doctor that will interpret their test as positive for Lyme.

Realize that everyone today will fail the Mount Sinai School of Medicine $4900 test for toxins. So let’s blame the neurotoxins and endocrine disruptors just like we blame the total body burden of infection, as properly tested everyone will have some Chlamydia or CMV or Coxackie or Candida and so on.

No one will pass the test at Harvard for bone lead levels. They have shown that the level in bone is in equilibrium with most other tissues in the body including the eye so there is a direct correlation with how high lead in bones is and how soon you develop a cataract. So there is no one on earth that does not need some lead out and since Lead makes Mercury as much as 100 times more toxic, who needs tons of tests to know what to do in most of the categories my F.I.G.H.T. program acronym represents.

So would it not be better medicine to offer some oral detoxification for the Heavy metals and the Toxins, with ZeoGold and BIOE’NR-G’Y C, Beyond Fiber, and some organic Greens and some Maca and help people eliminate suspect foods for a time. Before letting the outcome of the patient’s intervention with the doctor pass on the results of unreliable negative lab tests for Lyme, because of immune suppression until some treatment is started for awhile and then the test for Lyme often changes to positive. What a waste to not simply realize we are confronted with an epidemic of autoimmune diseases that has so many different presentations that over 100 conditions are now considered to be autoimmune related. These conditions deserve meaningful intervention and my F.I.G.H.T. program protects patients from Johnny One Note health care providers who focus only on one aspect of my program and thus only help a small percentage of patients.

Let’s broaden our approach and help everyone with empowering knowledge. Everyone we see today needs help to optimize every one of the categories in F.I.G.H.T. If we expand the FIGHT concept we would make F stand for FOCUS on positive thinking not just Food and H for hormones and Heavy metals and then really the G is not just Genetics but also the entire new field of Epigenetics where exposures to BISPHENOL A have led to overnight changes in Gene activation. They are permanent until treated with aggressive methylation support, as with the MSM and TMG found in BIOE’NR-G’Y C and the active forms of Folic Acid found in Beyond B12.

We all remember AIDS is acquired immune deficiency so now I recommend that this new epidemic just be renamed LOWERED IMMUNE METABOLIC ENCEPHALOPATHY SYNDROME or LIMES then we can start to be much more cost effective in improving the health of many who suffer without excess reliance on some lab test for Lyme related infections.

This link to MEDSCAPE may help broaden your knowledge regarding some aspects of this new epidemic. By putting LIMES category into a new AUTOIMMUNE RELATED condition it forces us to broaden our approach beyond antibiotics can help our patients who still will not be covered by insurance but at least they will not be turned away without receiving real help and we will not waste time with medical board fights. Patients will be taught something that I am confident for most will help them improve their health more than getting 6 months of IV antibiotics even if it were fully covered by their insurance company. It is not just an antibiotic deficiency we are encountering; read the book BEYOND ANTIBIOTICS!

It is like the old adage TEACH a man to fish or give him a fish; I prefer the teaching approach. Knowledge of what is really wrong with our health can be empowering but to put everything on one infection or one toxin and ignore leaky gut and food sensitivities, etc I feel  means we provide little long-term meaningful help to patients who deserve a broader understanding of what is really going wrong with their health.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com
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A Case of Ascending Paralysis: the Signs and Symptoms of Tick Paralysis
Menyoli Malafa, MSII; Veronica Tucci, JD, MS IV; Albert Vincent, PhD; Sajeel Chowdhary, MD
Posted: 03/26/2009; American Academy of Emergency Medicine.
2009;16(1):22, 26, 27 © 2009 American Academy of Emergency Medicine
http://www.medscape.com/viewarticle/589591

Summary
Tick paralysis (TP), a response to the neurotoxic effects of the salivary secretions produced by attached hard ticks (Ixodidae), is a syndrome that mimics a large number of better known neurological disorders. TP is a sporadic, seasonal, rural disorder in which acute ataxia often develops five to six days following a history of walking in grass or low brush, followed by ascending flaccid paralysis. Recognition and timely removal of the tick usually leads to complete resolution of symptoms, whereas continued feeding can lead to respiratory arrest and death. Follow-up includes species determination and patient surveillance for tick-borne infectious disease.

Discussion
TP is a worldwide disease, occurring in Australia, Europe, South Africa and throughout North America. In the United States, most cases occur in the Rocky Mountain states and the Pacific Northwest, including Washington, Montana, Oregon, Idaho, Wyoming, Nevada, Utah, Colorado and the northern parts of Arizona, New Mexico and California. However, cases have also been reported in central, southern and eastern states, including Texas, Oklahoma, Mississippi, Florida, Georgia, North Carolina, South Carolina, Virginia, Washington, D.C., Pennsylvania and New York. In Canada, most cases are encountered in the western part of the country, primarily southern British Columbia.[1,2] More than 60 species of ticks are known to cause paralysis, but only a handful are responsible for most cases. In North America, the disease is associated primarily with six species: Dermacentor andersoni (‘Rocky Mountain wood tick’), D. variabilis (‘American dog tick’), Amblyomma americanum (‘Lone Star tick’), A. maculatum (Gulf Coast tick), Ixodes scapularis (formerly I. dammini, ‘Blacklegged tick’) and I. pacificus (‘Western Black-legged tick’). Peak incidence occurs between April and June when nymphs and mature adults abound in low vegetation and climb upward, questing for their next host by extending their anterior pairs of legs.[1,3,4] Paralysis is a response to a neurotoxin secreted by the salivary glands of the arachnid.[1,5] The biochemistry and pharmacology of the specific paralysis- inducing toxins produced in North American ticks are yet to be fully elucidated, but current evidence points to a mechanism by which the toxins inhibit presynaptic acetylcholine release at the neuromuscular junction.[1,3,6] TP presents more often and more severely in children, suggesting a concentration-dependent relationship between toxin levels and symptom expression.[1,4] Signs and symptoms of TP begin about five to six days after the parasite has attached, when neurotoxin is secreted at its peak levels. These prodromal symptoms include restlessness, irritability, fatigue, nausea, paresthesias and possibly ataxia. Over the next 24-48 hours, the patient develops ascending symmetrical flaccid paralysis and weakness in the lower extremities. Over the course of the next day or two, paralysis and weakness may ascend to involve the trunk, axial and upper limb muscles. Cranial nerves may also become involved in an ascending pattern, resulting in bulbar, facial and/or extraocular paralysis. Patients demonstrate diminished or absent deep tendon and superficial reflexes while, aside from occasional paresthesias, their sensory exam remains normal. Pain and fever are absent. Death ensues following paralysis of the respiratory muscles.[1,5,7,8,9] Atypical presentations reflect variations in the site of tick attachment. There may be ataxia and associated cerebellar deficits without accompanying muscle weakness. The disorder may also present as an isolated facial paralysis without trunk or limb involvement. Another group of atypical presentations is unilateral paralysis and/or weakness, including isolated unilateral facial paralysis.[1,8] Tick paralysis is treated by removal of the tick. Although the site of attachment is most often the head and neck region, the entire body should be scrutinized, including ear canals, nostrils and genitalia. Multiple ticks should be suspected, and all must be removed.[1,4,7,10] Applications of petroleum jelly, nail polish, alcohol, a needle and heat are inappropriate. These measures may result in infection and cause the parasite to salivate or regurgitate more of its bodily fluids.
The tick should be grasped with blunt, angled forceps as close as possible to the skin and to the embedded mouthparts (hypostome). Wearing protective gloves, slowly pull the organism straight outward with a gentle and steady traction, without twisting its body. Do not burst the tick. The hypostome is usually deeply and firmly embedded and should be removed surgically should it come detached. Antiseptic solution is then applied to the wound, and the recovered tick and severed mouthparts may be preserved in 75% ethanol for identification. The patient should be instructed to return in the event of additional illness and educated on protective measures against ticks.
The symptoms of TP, at least those caused by North American species, typically resolve rapidly following removal of all ticks from the patient. Improvement in the condition of the patient subsequent to tick removal is confirmatory for the diagnosis. Species found in some other parts of the world, notably Ixodes holocyclus of Australia, produce a very potent neurotoxin and symptoms may not subside as quickly, even worsening after removal.[5] The prognosis depends on clinical presentation prior to removal. If all ticks were removed prior to the onset of bulbar weakness, the patient often makes a full recovery within the first 24 hours. However, if onset of bulbar symptoms occurs during continued feeding, the likelihood of fatal respiratory paralysis increases to 10%. Therefore, prompt of diagnosis and tick removal are paramount.[1,5,7,8] Because ticks are both vectors and reservoirs for various infectious diseases, it is important to educate the patient about this added risk for possible concurrent illnesses. Table 1 displays the geographical location and infectious diseases associated with North American tick species which are also known to cause TP.[1,8,11,12]

References
1.Cunha BA, editor. Tickborne Infectious Diseases Diagnosis and Management. New York: M. Dekker; 2000.
2.Meier J, White J. Handbook of Clinical Toxicology of Animal Venoms and Poisons. STATE: CRC Press; 1995.
3.CDC. Tick paralysis – Washington. Morbidity and Mortality Weekly Report 1996; 45(16): 325-6.
4.Schmitt N, Bowmer EJ, Gregson JD. Tick paralysis in British Columbia. Can Med Assoc J 1969 Mar 1; 100(9): 417-21.
5.Meriggioli MN, Howard JF, Howard Jr. JF, Harper CM, Harper Jr. CM. Neuromuscular Junction Disorders: Diagnosis and Treatment. STATE: Informa Health Care; 2003.
6.Grattan-Smith PJ, Morris JG, Johnston HM, Yiannikas C, Malik R, Russel R, Ouvrier RA. Clinical and neurophysiological features of tick paralysis. Brain 1997 Nov;120(Pt 11):1975-87.
7.CDC. Tick paralysis – Colorado. Morbidity and Mortality Weekly Report 2006 Sep 1; 55(34): 933-5.
8.Knoop KJ, Stack LB, Storrow AB. Atlas of Emergency Medicine. STATE: McGraw-Hill Professional; 2002.
9.Biller J. Practical Neurology. STATE: Lippincott Williams and Wilkins; 2002.
10.Gammons M, Salam G. Tick removal. Am Fam Physician 2002 Aug 15; 66(4): 646.
11.Winn WC, Kineman EW, Allen SD, Janda WM, Schreckenberger PC,Procop GW, Woods GL. Koneman´s Color Atlas and Textbook of Diagnostic Microbiology. STATE: Lippincott Williams and Wilkins; 2005.
12.Sonenshine DE, Mather TN. Ecological Dynamics of Tick-borne Zoonoses. STATE: Oxford University Press US; 1994.
13.Greenberg BM. Clinical cases in neurology from John Hopkins. Case 2: acute ascending paralysis in a 4-year-old body. MedGenMed 2003 Apr 9; 5(2): 36.

WE ALL HAVE SOME CHRONIC INFECTIONS; now there is another major breakthrough to prove this.

My Fight program clearly is meant to help us educate our patients that achieving optimal health is a lifetime challenge. With increasing sophistication of lab sciences, we will find many more challenges in every one of my FIGHT categories but with the epidemic of people with chronic fatigue, you now have even more reason to want to learn about Oxidative therapies like OZONE/UVB/SILVER.

However, a unifying approach to enhance our bodies ability to deal with the multifactorial nature of any chronic disease should increase our interest in learning more about Energy medicine. This broad topic includes Homeopathy, Accupunture, Prayer, Microelectric Current therapy, Magnetic Healing, Oxygen, Hyperthermia and much of what is now called Alternative Medicine.

The exerpt below is just one line from the great overview of viral infections and XMRV that is found in Autism and Prostate Cancer and Chronic Fatigue. This area of research is all new this year but we can expect that someone will soon find many more fungi issues or parasites issues. Of course tying impaired health to our toxin load is just beginning to be seriously considered. Do not fail to look for a moment at this article, as when you tell patients they need to deal with the chronic infection component of their current symptom complex, many feel you are off the wall.

Unless they have heard of Candida or Chlamydia, or CMV, or Herpes, or Lyme, they are not attuned to the need to lower their total body burden of pathogens. No one needs to spend the money to chase down which of these infections they have, as no one will test negative for one or more of these infections if adequately tested. So testing for most patients is impractical except to get their attention as to why they must do something about the infection component of my FIGHT program if they are to achieve optimal health.

“XMRV (xenotropic murine leukemia virus-related virus) is strongly associated with chronic fatigue syndrome/ME. The earlier study published in the journal Science was a joint study by the Cleveland Clinic, the National Cancer Institute, and the Whittemore-Peterson Institute of the University of Nevada with Drs. Vincent Lombardi and Judy Mikovits as lead authors. Here the lead author of the NIH/Harvard/FDA study, Dr. Harvey Alter, noted in a press conference that he considered his study a confirmation of the earlier WPI study, even though they had detected different MLV-related viruses (MRVs), rather than only XMRV. There does seem to be a greater variety of MRVs in chronic fatigue syndrome/ME patients than first understood. The WPI’s original study also showed some evidence of additional MRVs.”
Read more: http://www.ageofautism.com/2010/09/my-wife-my-daughter-and-xmrv.html

Sincerely,

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com

Full article: http://bolenreport.com/feature_articles/feature_article072.htm

Excerpt:

Most people battling chronic Lyme disease think of the illness as an infection caused by a bacterium known commonly as Borrelia Burgdorferi, generally transmitted via the bite of an infected tick.  What many don’t recognize, however, is that recovery from chronic Lyme disease requires a recognition that the disease is truly a much more complex illness.  Recovery often challenges one to consider more than just infection as the single causative agent involved in the disease process.  It is through looking beyond the infectious component of Lyme disease and understanding the equally important aspects of damaging heavy metals and other toxic insults that a more full and lasting recovery may be realized.

Garry F. Gordon MD, DO, MD (H) co-founded the American College for Advancement in Medicine (ACAM) and serves as the President of Gordon Research Institute.  Dr. Gordon graciously spent a couple of hours with me sharing his views on chronic Lyme disease and those factors that are important in recovering from chronic illness. 

Dr. Gordon acknowledges Lyme disease as a serious infection which can lead to a wide-variety of health challenges.  He does not, however, hyperfocus on the specific tick-borne pathogens which cause the disease.  He instead believes that a multitude of infections are prevalent in anyone with chronic ill health.  In addition to these numerous infections, our state of health is closely tied to our total body burden of endogenous and exogenous toxins.  When looking at why illness is present, it is important to look at a number of factors including genetics, chronic infections, and total body burden of heavy metals and other toxins.

Peering into one’s genetic makeup can be quite helpful when establishing the proper course of action and considering what factors may have contributed to one’s state of health.  The more precisely a practitioner can understand the genetic contributors, the more accurately a treatment protocol can be outlined to fit a person’s unique needs.  As an example, a specific gene mutation can suggest an inability of the body to remove toxic heavy metals.  Thus, even tests performed to determine whether or not one is heavy metal toxic can be incorrect if the metals are not being released due to this specific genetic profile.  Where many doctors may miss a heavy metal toxicity issue in these patients, a practitioner incorporating a genetic review into their diagnostic workup is much better equipped to evaluate the potential impact of toxic metals on the overall state of health.

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The Vitamin D Newsletter
Another Autism Case Report
by John Cannell, M.D.
vitamindcouncil@vitamindcouncil.org

January 30, 2010.
(This is a periodic newsletter from the Vitamin D Council, a non-profit trying to end the epidemic of vitamin D deficiency. If you would like to subscribe please go to the Vitamin D Council’s website. http://www.vitamindcouncil.org/)
This month, I dedicate the entire newsletter to a mother’s lengthy case report of her autistic son. Other than name and place of residence, the letter was not edited.
Dear Dr. Cannell:
At age 2.5 years, between December 2007 and January 2008, my son experienced a fairly dramatic onset of symptoms that led to his diagnosis of autism. His symptoms (many of which we did not even know the terminology for at the time they first occurred) included:
–The inability to sleep at night, we would put him to bed at 8:00 or 8:30 p.m. following his normal bedtime routine
–Development of anxiety and refusal to leave the house even to do preferred activities
–Obsessive-repetitive questions and monologuing/run-on speech
–Sensory issues (refusal to wear jeans or any fabrics other than fleece, screaming hysterically at bath time, complaining and covering eyes in sunlight, covering ears for everyday noises that had not bothered him before (toilets flushing, pulling pots and pans from cupboards, etc.)
–Toe-walking
–Flapping and self-stimulating behaviors (repeatedly tapping his cheeks and eyes with all ten fingers, continually twisting up his fingers in pretzel-like configurations, holding objects in his peripheral range of vision and straining to see them from the corner of his eyes)
–Development of an unusual pattern of stuttering/vocal tic at the end of words,he would repeat the last sound/syllable,”I don’t want to go to the store-or-or-or-or-or-or. It won’t be fun-n-n-n-n-n-n-n.” He would make sounds even in his sleep “n-n-n-n-n-n” or “s-s-s-s-s-s-s”
–Loss of muscle tone (stopped walking up and down stairs and began crawling/sliding instead, decline in balance and motor skills)
–loss of handedness (began switching left to right hand, after seeming predominantly left-handed)
–Marked increase in hyperactivity
–Frequent spacing out/unresponsive episodes
Our son and his twin sister were born at 36 weeks, 5 days on March 17, 2005 after four months of bed-rest. As early as their 8 week appointment, I mentioned to our pediatrician that we had concerns about our son’s eye contact and social responsiveness (in comparison to his sister). I felt that I was having more difficulty bonding with him. We were told “don’t worry, but don’t wait” and were referred to our state’s Early On intervention program. At the end of June a physical therapist and speech pathologist from our intermediate school district came to our home to evaluate our then 3 month old son and told me that he was doing just fine and that I was worrying too much. I agreed that by the time they saw him he had begun smiling and making better eye contact.
We didn’t worry again about our son until fall 2006. He had walked just before his first birthday, but by 18 months+ he still seemed clumsy and prone to falling compared to his sister. We took him back to the intermediate school district for evaluation and were told that all of his development seemed to be in the normal range and that we shouldn’t worry. We were advised that we could take him to music and gym classes to work on his coordination and told that we could pay for private physical therapy if we elected. We followed all of the recommendations.
For a year, we didn’t notice any other changes until the sudden onset of symptoms listed above when he was 2.5 years. With the sudden onset of symptoms above, we took our son to see a number of specialists during the winter of 2008 including a neurologist (who diagnosed him with Asperger Syndrome), a psychologist (who diagnosed with autism), and a second psychologist who specialized in the treatment of autism (who diagnosed him with Pervasive Developmental Disorder Not-Otherwise-Specified). All three diagnoses are on the autism spectrum. He also began seeing an occupational therapist, a speech therapist, a behavioral specialist, and a DAN! (Defeat Autism Now!) doctor for dietary interventions. We saw a dramatic improvement by April/May of that year. Nearly all the symptoms on the list above had resolved. We assumed the improvements were due to diet but he started to go into the sun around that time. Our son slept well and spent many peaceful, happy and anxiety-free months during the spring and summer after turning three.
In mid-November 2008, I sent the following e-mail to the DAN doctor who had been helping us with our son.
“You saw our son Jonathan Switzer a few times regarding his autism diagnosis and diet issues, etc. He had a regressive period last winter from about December through April when his autism was diagnosed, then did pretty well all summer. Nursery school started off okay, too, but now he seems to be having another regression.
Main symptoms:
–Great difficulty getting to sleep (fidgets for 2 plus hours most nights while he had been falling asleep easily for several months prior to that)
–Marked increase in anxiety (again refusing to leave the house even to do things he loves, frequently shaking/clenching and telling us “I’m scared)
–Onset of OCD-like behaviors (afraid to get hands dirty, get extremely upset if he gets even tiny drips of water on himself)
–Increase in self-stimulatory behaviors (flapping, fidgeting, noise-making)
–Frequent crying jags and telling us he’s just giving up on everything
We have had other parents tell us that their kids on the spectrum have a worsening of symptoms during the winter months and we feel like we are observing this same pattern. We’ve done some reading about light therapy for depression/anxiety and to help correct disturbed sleep patterns and would like to give it a try for Jonathan.
Wondering if you have ever prescribed a light therapy box for pediatric patients before. Our insurance told us they will cover it with a diagnosis of Seasonal Affective Disorder, but I don’t even know if that is something that can be diagnosed in children. Guess we’re willing to try anything at this point. Do you know much about this type of therapy?”
Neither the DAN Doctor nor our pediatrician would write a prescription for a therapy light, so we purchased one on our own and found it made no discernible impact on his symptoms.
By December, our son’s symptoms had worsened further and we decided to put him in a very expensive and intensive autism treatment program through our local hospital. He made slow progress during his participation in the program from January through April. He was also involved in speech and occupational therapy during the winter months. At his IEPC meeting at school in March, we were encouraged to put him in the district’s program for children with developmental delays. We instead elected to register him for regular pre-school for the following year.
During that winter, I was crying to some friends about my son and describing his seemingly seasonal pattern of symptoms. We had just seen a second neurologist searching for help, and I was extremely frustrated when, after listening to my son’s symptoms and history, he told me bluntly, “There is nothing seasonal about autism,” then suggested that we put our son on an anti-depressant. We refused the medication. One of the friends I was crying to is a research librarian and the other is a medical researcher. After our conversation, they located and e-mailed me a few journal articles they thought might help, one of the articles was by Dr. Cannell and discussed his vitamin D theory of autism. Reading the article was one of those “Aha!” moments and I felt hopeful that Dr. Cannell was on to something.
By June our son was released from both speech therapy and occupational therapy and we were told that he no longer showed any delays for his age. When he had begun occupational therapy in January, the OT had been astonished at our son’s lack of muscle tone. She recommended that he also receive Physical Therapy services, so we went on a long waiting list. Our initial OT was in a car accident, and in May we were transferred to a new OT. When the new OT first saw our son, she said could not believe he was the same child described in the notes. By May the low muscle tone, hyperactivity and distractibility noted in his file, were no longer evident. His turn came up for physical therapy and we were told he no longer needed it.
Our son has always spent a lot of time outdoors in the summer, without sunblock. He had a happy and relaxing summer. As fall/back-to-school approached, I began to fear the onset of another regression and again read the article by Dr. Cannell my friend had sent. I visited his website and decided we would try a vitamin D supplement. Our pediatrician did not encourage any dose higher than 400 i.u. (that found in a typical multivitamin) but did write a script to have his 25-hydroxy level tested. In August his level was 37, so we started him on 5,000 iu daily and had his level retested on October 21st. By October his level was 96. The pediatrician was concerned that this was too high and told us he should not have more than 400 iu per day.
Knowing that Nov-March are typically his worst months, we reduced the dosage down only to 3,000 iu from October through mid-December. At an appointment in December our son was doing wonderfully (none of his usual fall/winter symptoms yet evident) and the pediatrician told us 3,000 iu was too much and that we should be giving no more than 400 iu. In mid-December we reduced the dose to 1,500 iu. By the beginning of January we noted a marked loss of eye contact. We also noted that our son was again interchanging his right hand for writing and eating (after using his left hand exclusively for 8+ months). We increased his vitamin D level to 4,000 iu daily in early January. On January 11 we had his 25-Hydroxy level checked on January 11 and found that it was 89. By the end of January, we and his grandparents noted improvement in his eye contact.
In January 2010 we attended his preschool conferences. The teacher had marked cards with the following code (1=age appropriate, 2=developing, 3=area of concern). Our son received 1s in all areas with the exception of hopping on one foot and balance beam where he received 2s. We were told that he is on par with or ahead of his peers in all areas (academic, fine motor, etc.), and that his teacher had noted no unusual symptoms or concerns.
During the fall/winter 2009-2010 our son has been free from nearly all of the most troubling symptoms that plagued him the previous two winters. The following example may demonstrate the improvement in his daily life since last winter.
One of our son’s low points was a Christmas party we attended in December 2008. Before leaving the house to attend the party our son screamed and yelled about having to take a bath and because we would not let him wear sweatpants to the party. He then begged us not to make him leave the house. During the 40 minute trip to the party our son asked us repetitive questions and talked incessantly. Upon arriving at the party, he immediately walked into an unoccupied room adjacent to the room where the party was occurring, and put his face into the corner. Despite much coaxing by my husband and me, he refused to come out of the corner.
After approximately 45 minutes of standing in the corner we managed to get him out through the promise of some food rewards. He proceeded to walk around and around the perimeter of the living room where all of the other kids were playing. He rubbed himself along the walls and covered his ears as he walked. He finally settled into playing alone in a corner of the room. All of the kids at the party participated in a book exchange. Our son refused to come to the area where the other kids were gathered. We coaxed him over only to have him throw the book he received and refuse to thank the parent who had purchased it for him. He spent much of the evening in time-outs for that and other inappropriate behavior.
In June of 2008, after playing in the sun for several months, we met for a picnic with the same group of friends at a local park. Our son ran up to the other children and joined right in playing bulldozers in the sand with them. He behaved and interacted in a completely appropriate and typical way during the picnic which lasted several hours.
This year (2009) we attended the same Christmas party at the same house. Our son got ready and left for the party without anxiety or incident. He chatted normally during the drive to the party. He walked into the house, said, “Hey, check out my new train,” to some of the kids already playing and settled in to playing happily with the other kids. During the book exchange, he received a book, smiled and gave a big hug to the person who gave it to him.
In December of 2008, I took a leave from my job so I could get my son to the intensive behavioral treatment program he was in and to all of his other therapy appointments. I dedicated 40-60 hours per week to my son’s various appointments and home therapy program.
This winter (January 2010), a former colleague asked me what Jonathan’s current therapy program consists of. I told her I spend about 30 seconds each day opening the jar of vitamins and giving him his chewable vitamin D. In my opinion, the 3 minutes or so I spend each week giving him his vitamin D have been much more effective, and much less expensive, than any other treatment we have pursued.
Thank you.
Jeannette, Wisconsin
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~
Dear Jeanette:
You’re welcome. Several things need comment. First, the symptoms are typical of autism. Second, the seasonality of symptoms suggest a vitamin D deficient disease. Third, the treatment in the spring of 2008 seemed effective but, in hindsight, it was simply due to spring sun exposure. Fourth, as you may now know, light boxes for seasonal affective disorder make no vitamin D. Fifth, your pediatrician knows little about Vitamin D other than what committees tell him; your decision to ignore his advice probably saved your son’s brain from further injury, as autism is a progressive inflammatory destruction of brain tissue. Sixth, the fact that you needed bed rest and gave birth prematurely suggests you were Vitamin D deficient during your pregnancy.
Seventh, his twin sister has never had autism, despite the same intrauterine environment. This is consistent with my theory, that autism is caused from a quantitative, not qualitative, variation is one of the enzymes that metabolize Vitamin D. That is, there are no structural differences in these enzymes in autism, only a genetically determined difference in the amount present. These enzymes are responsive to estrogen; estrogen protects the brain from being damaged by low Vitamin D, probably by increasing the amount of activated Vitamin D present, explaining why boys are four times more likely to have the disease.
The report that your son deteriorated when his dose was reduced from 3,000 to 1,500 IU suggests autistic children need adult doses of Vitamin D. When you reduced the dose from 3,000 to 1,500 IU/day he worsened although his level on 1,500 IU/day was probably still greater than 50 ng/ml. This makes me think that dosage needs to be stable and suggests that Professor Reinhold Vieth’s theory of a detrimental seasonal resetting of the intercellular metabolism of Vitamin D may even be true at levels above 50 ng/ml, where the body is storing the parent compound, cholecalciferol, in muscle and fat.
His current dose of 4,000 IU per day is perfectly safe and will give him a level of 80-100 ng/ml, inside the reference ranges of American laboratories. Toxicity (asymptomatic high blood calcium) begins somewhere above 200 ng/ml. Generally speaking, autistic children should take 2,000 IU per every 25 pounds of body weight for six weeks, then have a 25(OH)D blood test and adjust the dosage to get into the high end of the reference range, 80-100 ng/ml.
Although I first published the Vitamin D theory of autism theory 3 years ago, few autistic children are currently treated for their Vitamin D deficiency. This is due to several reasons. One, those who think, correctly, that autism is a genetic disease, stop thinking after that, reasoning that genetic diseases are untreatable. Such thinkers do not understand epigenetics (upon the genome). Vitamin D is probably the heart of epigenetics, as nothing works upon the genome like vitamin D.
Secondly, the “all autism is caused from vaccinations” crowd cannot accept the Vitamin D possibility as it threatens their core beliefs. They simply cannot change their minds.
Finally, as you now know, organized medicine would say you should stop the vitamin D and watch your son deteriorate, which is why slavery to evidence based medicine is fine for scientists and unethical for practitioners.
John Cannell, MD
Executive Director
Vitamin D Council
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San Luis Obispo, CA 93401

I am quite sure that this book by Dr Schaller is one you will want access to if you are thinking of incorporating my F.I.G.H.T. Program in your practice.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

FREE 2009 UPDATED BABESIA TEXTBOOK

http://ebooks.hopeacademic.com/BabesiaUpdate2009-Ebook.pdf

Dr. Schaller has decided limiting access to this new book due to cost, despite the immense cost and years of reading and research to write it, is the wrong approach.

Books on super specific issues do not sell and do not make money to even remotely break even.

The reasonable money being made in good sales is certainly not worth one life. It is not worth one person becoming disabled and non-functional for years or decades.

http://ebooks.hopeacademic.com/BabesiaUpdate2009-Ebook.pdf

BASED ON YEARS OF RESEARCH, STUDY OF VIRTUALLY ALL MAJOR PUBLISHED SCIENTIFIC/MEDICAL LITERATURE

TREATMENTS BASED ON INHERITED TREATMENT FAILURES

NEW LAB TESTING OPTIONS WHICH MIGHT CATCH THE CAUSE OF LOST PROGRESS OR RELAPSE

THE COPYRIGHT STAYS IN PLACE BUT IS NOT VIOLATED IF YOU USE FOR YOUR OWN PERSONAL USE. FEEL FREE TO PRINT THE BOOK.

IF YOU DO NOT SELL IT, YOU CAN GIVE IT TO ANYONE FOR FREE. IF YOU DO NOT SELL THE BOOK, IT CAN BE FORWARDED WITHOUT LIMIT.

YOU cannot alter the text in any manner.

Babesia can cause death, disability, obesity, serious fatigue, migraine torture and a hundred other things it needs to be free to everyone.

We also hope it will help some interested healers catch stealth Babesia more often because we feel it is common, and not an occasional finding in a Lyme positive patient. Many new Babesia species are being found in humans even in the last four years, so the list of human Babesia species and their variants is not finalized

Finally, the cost for top laboratories to keep up and have perfect diagnostic testing is an unrealistic expectation, so other new ideas and included to help show low levels of Babesia that have significant body effects based on solid research articles.

PLEASE DO NOT SELF TREAT BASED ON THIS BOOK.

NO INFORMATION IS MEANT TO BE AUTHORITATIVE AND ALL CARE IS UNDER THE SUPERVISION OF A LICENSED MEDICAL WORKER.

Be Better!! Be Well!

Rona. C
Office Manager

Pregnant women do not need mercury injected into their bodies. Learn the truth in the  book  “The AutoImmune Epidemic “ by Donna Jackson Nakazawa who twice suffered  total body paralysis following  flu shots  ( Guillian Barre)  and who now points out that there are serious genetic changes  in our population so that one size fits all medicine is dangerous .

Dr Sherri Tenpenny, who has gathered more information on vaccines than almost anyone alive, has prepared some useful information for you to hand out to patients if you should choose.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

Hello Everyone,
I put the following information together to help all pregnant women know the Truth about the Flu Shot for pregnancy.

A pdf will also be posted on www.drtenpenny.com so you can print this for family, friends and patients.

Best regards,
Dr Sherri Tenpenny
www.DrTenpenny.com

The Truth about Flu Shots in Pregnancy
Ø  Recommendations for vaccinating pregnant women are new:
•         2004: The Advisory Committee on Immunization Practice (ACIP) of the CDC began recommending vaccination in all pregnant women regardless of trimester. By vaccinating everyone, researchers estimated that an average of 1 or 2 hospitalizations could be prevented for every 1,000 pregnant women vaccinated.
o    REF: MMWR: May 28, 2004 / 53(RR06);1-40.
•         2009:  There is insufficient evidence to recommend routine flu shots as the standard of practice for healthy women beginning in early pregnancy.
o   REF: Skowronski  DM, De Serres G.  Is routine influenza immunization warranted in early pregnancy? Vaccine. Jul 30;27(35):4754-70. 2009.

Ø Pregnant woman are not frequently hospitalized for flu:
•         1998: Hospitalization ranged from 3.1 per 10,000 women-months in the first trimester to 10.5 per 10,000 in the third trimester.
o    REF: Neuzil KM, et al. Impact of influenza on acute cardiopulmonary   hospitalizations in pregnant women.  Am J Epidemiol 1998;148:1094-1102.
•         2004: Women in the third trimester were hospitalized at a rate (250/100,000 pregnant women), a rate comparable to non-pregnant women who had high-risk medical conditions. Consider that malpractice liability may play a role in the decision to admit third trimester women to the hospital.
o   REF: MMWR: May 28, 2004 / 53(RR06);1-40.
•         2009: The numbers of pregnant women hospitalized with pandemic H1N1 infection are small. Pregnant cases represented 34 of 5,469 H1N1 cases diagnosed during a four month study.
o    REF: Jamieson DJ, et al. H1N1 2009 influenza virus infection during pregnancy in the USA. Lancet. Aug 8;374(9688):451-8. 2009.

Ø  Vaccinating pregnant women has not reduced hospitalizations.
•         1997-2002:  A study of 49,585 pregnant women there was no statistically significant difference in rates of illness among vaccinated vs. unvaccinated women (4.5/10,000 vs.4.4/10,000). There were only two admissions per season for pneumonia.
o    REF: Black SB, et al. Effectiveness of the influenza vaccine during pregnancy in preventing hospitalizations and outpatient visits for respiratory illness in pregnant women and their infants. Am J Perinatol 21:333-339. 2004.

Ø  Pregnant women are generally as healthy as non-pregnant women.
•         1997-2002:  A study of 49,585 pregnant women, only 4.7% had outpatient visits for influenza-like illness.
o    REF: Black SB, et al. Effectiveness of the influenza vaccine during pregnancy in preventing hospitalizations and outpatient visits for respiratory illness in pregnant women and their infants. Am J Perinatol 21:333-339. 2004.
•         2009: Symptoms of H1N1 influenza infection are similar in both pregnant and non-pregnant general population except some pregnant women have more shortness of breath.
o    REF: Jamieson DJ, et al. H1N1 2009 influenza virus infection during pregnancy in the USA. Lancet. Aug 8;374(9688):451-8. 2009.

Ø Careful examination of the numbers used to mass vaccinate pregnant women reveals:
•         H1N1 occurs infrequently in pregnant women: 34 cases among an estimated 3,392,000 pregnant women in the U.S.
•         Among the 5,469 confirmed cases in the study, 0.62% were in pregnant women (34/5,469)
•         Claims that hospital admissions among pregnant women are much higher than in the general population are based on the use of numbers that accentuate the effect  (11/34 vs. 229/5,469).
•         The admission rate for pregnant women was 0.32 per 100, 000 and the estimated admission rate in the general population was 0.076 per 100 000. Hence the statement, “Pregnant women were more than four times more likely to be admitted than was the general population.”
o    REF: Jamieson DJ, et al. H1N1 2009 influenza virus infection during pregnancy in the USA. Lancet. Aug 8;374(9688):451-8. 2009.

Ø Both seasonal flu shots and 2009 H1N1 flu shots are labeled “Pregnancy Category C” drugs, meaning:
•         Animal reproduction studies have not been conducted.
•         It is not known whether influenza vaccines can cause harm to the fetus.
•         It is not know whether influenza vaccines can affect reproduction capacity.
•         It is not known whether influenza viruses from vaccines are excreted in human milk.
o    REF: From a review of all manufacturer package inserts

Ø Are anti-viral drugs safe to take while pregnant?
•         Both Tamiflu (oseltamivir) and Relenza (zanamivir) are classified as “Pregnancy Category C” drugs, meaning, there is insufficient information to assess potential risks to the fetus.
•         In lactating rats, Tamiflu was excreted in the milk. It is not known whether Tamiflu is excreted in human milk.
o    REF: From the package inserts

Info complied by Dr. Sherri Tenpenny at www.DrTenpenny.com

www.PandemicFluOnline.com www.MedicalVoices.org

This is very interesting research that would cause you to want to take exposure to flu more seriously!  First, let’s stay healthy so we do not get flu or colds with all that I discuss from Immuni-T 2 and 3 to long term detoxification, as in FIGHT program. Then since for many that is too rigorous a requirement, so let’s have an EMERGENCY SUPPORT PACKAGE in the homes of anyone that realizes who important this new research is!  Have BioE’nR-G’y C, ACS 200, and high dose D AND A available to immediately begin at the first sign of active infections.

What if their vaccines turn out to be as wrong for stopping H1N1, as MMR has been shown to be for compromised children? After MMR in autistic kids the live virus from the vaccination later has been shown to be growing in the CSF of these children.  Will the current testing of the new swine flu vaccines even attempt to look for how many receiving the vaccines actually wind up being infected with the virus instead of being protected against the virus, as they are not able to launch an immune response to the vaccine?

It appears that the virus accesses the neuron through the axons in the GI tract and lung. This sets the stage then for loss of Dopamine secreting cells over time.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

Recent studies have suggested that the currently circulating strain of avian influenza has similar pathology to the 1918 flu. Though the subtypes of the viruses are different (Spanish flu shares the H1N1 subtype with the current H1N1 swine flu, whereas avian influenza has an H5N1 subtype), both viruses appear to enter the central nervous system (CNS) and can cause encephalitis, or inflammation of the brain.

Highly pathogenic H5N1 influenza virus can enter the central nervous system and induce neuroinflammation and neurodegeneration
http://www.pnas.org/content/early/2009/08/07/0900096106.abstract

1.       Haeman Janga,b,
2.       David Boltzc,
3.       Katharine Sturm-Ramirezc,1,
4.       Kennie R. Shepherda,2,
5.       Yun Jiaoa,
6.       Robert Websterc and
7.       Richard J. Smeynea,3

+Author Affiliations
1.       Departments of aDevelopmental Neurobiology and
2.       cInfectious Diseases/Virology, St. Jude Children’s Research Hospital,
262 Danny Thomas Place, Memphis, TN 38105-3678; and
3.       bIntegrated Program in Biomedical Sciences, University of Tennessee
Health Science Center, Memphis, TN 38163
1.      1Present address: Fogarty International Center, National Institutes
of   Health, 16 Center Drive, Room 202, Bethesda, MD 20892.
2.      2Present address: Department of Environmental and Occupational
Health, Rollins School of Public Health and Center for
neurodegenerative Disease, Emory University, Whitehead Biomedical
Research Building, 5th Floor, Room 575.1, Atlanta, GA, 30322.

Abstract
One of the greatest influenza pandemic threats at this time is posed by the highly pathogenic H5N1 avian influenza viruses. To date, 61% of the 433 known human cases of H5N1 infection have proved fatal. Animals infected by H5N1 viruses have demonstrated acute neurological signs ranging from mild encephalitis to motor disturbances to coma. However, no studies have examined the longer-term neurologic consequences of H5N1 infection among surviving hosts. Using the C57BL/6J mouse, a mouse strain that can be infected by the A/Vietnam/1203/04 H5N1 virus without adaptation, we show that this virus travels from the peripheral nervous system into the CNS to higher levels of the neuroaxis. In regions infected by H5N1 virus, we observe activation of microglia and alpha-synuclein phosphorylation and aggregation that persists long after resolution of the infection. We also observe a significant loss of dopaminergic neurons in the substantia nigra pars compacta 60 days after infection. Our results suggest that a pandemic H5N1 pathogen, or other neurotropic influenza virus, could initiate CNS dis

Why is FDA so afraid to alert consumers to the possibility that amalgams contribute to total body mercury levels and that combined with eating fish, which they refuse to require adequate labeling there also, means that consumers have no knowledge why they have so many health issues in their family.

It seems that the experts will disagree, as to how much mercury in our body is from coal burning for power plants that settles in oceans and comes up in the food chain vs. how much is in the particulates we breath, which in one study in San Francisco was shown to be over 30% of all the total body burden of mercury. Breathing particulates carrying mercury is the major contributor or merely the second largest contributor may vary from research paper to research paper and be somewhat related to environmental circumstances of the patient group being studied.

We are very confident, however, that there is no safe level for lead or mercury and that there are serious synergies in their toxic effects on our health. It will come to pass someday that patients will know how much is in vaccines, amalgams, fish, and air they breathe and or water they bath in and in all of the many contributors to our body burdens of lead and mercury, which becomes complex as when we are born we are already loaded with them so there is no easy way out.

So I believe the day will come that everyone will consume oral chelators or improved Zeolite products daily and they will be as available as salt and pepper wherever we eat.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

FDA’s Mercury Ruling Defies ALL Scientific Reasoning
by Dr. Mercola
August 22 2009
http://articles.mercola.com/sites/articles/archive/2009/08/22/FDA-has-the-Audacity-to-Claim-Mercury-is-Completely-Harmless.aspx

In the video above I speak with Charles Brown, legal counsel for the Consumers for Dental Choice, which is a nonprofit corporation whose purpose is to educate the public about the health and environmental dangers of mercury fillings, and to ensure more effective government oversight on amalgam. Charles discusses the processes he’s been undertaking for the last 10 years to get dangerous mercury fillings removed from the market, and brings you up to speed on where we are today with the FDA’s most recent, atrocious ruling.
The U.S. FDA has issued a final regulation classifying dental amalgam without calling for stringent precautions for pregnant women and children — even though last June a court settlement filed by the Consumers for Dental Choice required the FDA to withdraw claims of mercury amalgam’s safety from its Web site and issue an advisory indicating:
“Dental amalgams contain mercury, which may have neurotoxic effects on the nervous systems of developing children and fetuses.”
Instead, the FDA has classified the fillings as class II devices, meaning the agency is claiming that they are completely harmless. This stands in direct contradiction of the conclusions of the FDA’s own panel of scientific experts, and the findings of the International Academy of Oral Medicine and Toxicology (IAOMT).
In fact, mercury dental fillings contribute 2 to 3 times as much mercury to the human body as all dietary and environmental sources combined. IAOMT is urging the FDA to change the ruling, ban dental amalgam from commerce and issue a mandatory recall on the product.

Charles Brown says:
“FDA broke its contract and broke its word that it would put warnings for children and unborn children for neurological damage. Bowing to the dental products industry, FDA for the first time in its history pulled a warning about neurological harm to children.”
“This contemptuous attitude toward children and the unborn will not go unanswered,” said Brown.  “We will see FDA in court.”
Vapors from dental mercury go into the human body. Due to mercury waste, amalgam is also increasingly targeted by environmentalists. Amalgam has also become controversial because the middle-class has largely moved to non-toxic alternatives while the poor, minorities, and institutional recipients, such as soldiers and prisoners, still get mercury amalgam.

Sources:

Medical News Today July 29, 2009

International Academy of Oral Medicine & Toxicology Press Release (PDF)

International Academy of Oral Medicine & Toxicology Position Paper on Dental Amalgam (PDF)