Bisphenol A – Comments from Dr. Gordon and Research Paper
By Linda on Sep 14, 2010 in Toxins | comments(0)
Toxins work in many ways. We can now better understand the causes behind our epidemic of obesity and diabetes and impaired suboptimal health, as a nation. The attached research will show that BPA (BISPHENOL A) is worse than any of us realized yet the lie goes on and, until recently, FDA still insisted it and many other toxins are perfectly safe.
Read this from Harvard and NIH and I hope that this knowledge will start you on the road to lifelong detoxification. We are all living longer, but not better, unless you start to detox daily for life. This is just one of the many toxins that are preventing you and your patients from enjoying optimal health. It is behind the EPIGENETIC changes that impair methylation, as documented by Randy Jirtle at Duke that shows the obesity and diabetes that starts when Agouti mice are exposed to BPA and continues forever in their offspring unless you aggressively provide methylation support.
So you need all the CH3 ( Methyl) support you can get and I have built into Beyond B12 three forms of Folic acid along with methyl cobalamin. Also the world’s most advanced Vitamin C delivery system BioEn’R-G’y C provides additional methylation support with its MSM and TMG (Trimethylglycine).
These are just some of the reasons my FIGHT program so far helps most patients even when other nutrient based programs had failed. The devil is in the details and this research paper should make you wake up to the fact that you have this and phthalates in heavy amounts in you and your patients and your children born and yet to be born. It is too late to escape so plan to start Zeolite, high dose C and D and Fiber with MACA and Organic Greens based treatment that really works. Why not try my “Power Drink” approach using those ingredients and add one ZeoGold premeasured dose in water for a month then decide if it is worth it to you to feel and function that much better! Nothing works optimally until we neutralize the toxins including mercury and lead and all of the organic toxins starting with BISPHENOL A.
Attached is entire paper on In vitro molecular mechanisms of bisphenol A action.
Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com
Excerpt:
Bisphenol A (BPA, 2,2-bis (4-hydroxyphenyl) propane; CAS# 80-05-7) is a chemical used primarily in the manufacture of polycarbonate plastic, epoxy resins and as a non-polymer additive to other plastics. Recent evidence has demonstrated that human and wildlife populations are exposed to levels of BPA which cause adverse reproductive and developmental effects in a number of different wildlife species and laboratory animal models. However, there are major uncertainties surrounding the spectrum of BPA’s mechanisms of action, the tissue-specific impacts of exposures, and the critical windows of susceptibility during which target tissues are sensitive to BPA exposures. As a foundation to address some of those uncertainties, this review was prepared by the “In vitro” expert sub-panel assembled during the “Bisphenol A: An Examination of the Relevance of Ecological, In vitro and Laboratory Animal Studies for Assessing Risks to Human Health” workshop held in Chapel Hill, NC, Nov 28-29, 2006.
The specific charge of this expert panel was to review and assess the strength of the published literature pertaining to the mechanisms of BPA action.
However, the primary endocrine disrupting activities of BPA extend beyond its ability to mimic, enhance or inhibit the activity of endogenous estrogens and/or disrupt estrogen nuclear hormone receptor action, and include the following: effects upon the androgen systems [22–29]; disruption of thyroid hormone function [30–40]; diverse influences on development, differentiation and function of the central nervous system [40–46]; and influences on the immune system [21,47–65]. There is additional clear evidence for BPA to directly impact intracellular signal transduction pathways through mechanisms independent of the transactivational activity of nuclear hormone receptors [43,46,66–79]. Secondary metabolic and pharmacokinetic actions of BPA that impact its bioavailability and bioavailability of endogenous steroid hormones have also been described.
