Lyme is all around us but I believe we will help many more if we give up on blaming everything on the tick related introduction of more pathogens than we had the day before we are bit.  It is confusing to people, as too often Lyme tests are inconclusive. So let’s rename the condition LIMES (Lowered Immune Metabolic Encephalopathy Syndrome) or LIMNS (for Neuropathy, as in MS like conditions) or LIMAS (Arthropathy when it is more arthritic in presentation), as these names move us closer to seeing the true picture.

It is sad to turn patients away with these devastating symptoms when the Infectious Disease Association guidelines force us to say it is Lyme. I am certain broadening the approach to Food sensitivities, other Infections, Genetics, Heavy metals and Hormones and Toxins would wind up with better results than the low batting average that is reported from long-term IV antibiotics, which are often reported as low as 33% about which Lyme critics point out is the response rate to placebos. If we focus on my F.I.G.H.T. program and do something to help deal with the obvious issues that can be found in almost anyone in any of these categories, we can be more cost effective and actually help more patients, as they will stop looking just for a doctor that will interpret their test as positive for Lyme.

Realize that everyone today will fail the Mount Sinai School of Medicine $4900 test for toxins. So let’s blame the neurotoxins and endocrine disruptors just like we blame the total body burden of infection, as properly tested everyone will have some Chlamydia or CMV or Coxackie or Candida and so on.

No one will pass the test at Harvard for bone lead levels. They have shown that the level in bone is in equilibrium with most other tissues in the body including the eye so there is a direct correlation with how high lead in bones is and how soon you develop a cataract. So there is no one on earth that does not need some lead out and since Lead makes Mercury as much as 100 times more toxic, who needs tons of tests to know what to do in most of the categories my F.I.G.H.T. program acronym represents.

So would it not be better medicine to offer some oral detoxification for the Heavy metals and the Toxins, with ZeoGold and BIOE’NR-G’Y C, Beyond Fiber, and some organic Greens and some Maca and help people eliminate suspect foods for a time. Before letting the outcome of the patient’s intervention with the doctor pass on the results of unreliable negative lab tests for Lyme, because of immune suppression until some treatment is started for awhile and then the test for Lyme often changes to positive. What a waste to not simply realize we are confronted with an epidemic of autoimmune diseases that has so many different presentations that over 100 conditions are now considered to be autoimmune related. These conditions deserve meaningful intervention and my F.I.G.H.T. program protects patients from Johnny One Note health care providers who focus only on one aspect of my program and thus only help a small percentage of patients.

Let’s broaden our approach and help everyone with empowering knowledge. Everyone we see today needs help to optimize every one of the categories in F.I.G.H.T. If we expand the FIGHT concept we would make F stand for FOCUS on positive thinking not just Food and H for hormones and Heavy metals and then really the G is not just Genetics but also the entire new field of Epigenetics where exposures to BISPHENOL A have led to overnight changes in Gene activation. They are permanent until treated with aggressive methylation support, as with the MSM and TMG found in BIOE’NR-G’Y C and the active forms of Folic Acid found in Beyond B12.

We all remember AIDS is acquired immune deficiency so now I recommend that this new epidemic just be renamed LOWERED IMMUNE METABOLIC ENCEPHALOPATHY SYNDROME or LIMES then we can start to be much more cost effective in improving the health of many who suffer without excess reliance on some lab test for Lyme related infections.

This link to MEDSCAPE may help broaden your knowledge regarding some aspects of this new epidemic. By putting LIMES category into a new AUTOIMMUNE RELATED condition it forces us to broaden our approach beyond antibiotics can help our patients who still will not be covered by insurance but at least they will not be turned away without receiving real help and we will not waste time with medical board fights. Patients will be taught something that I am confident for most will help them improve their health more than getting 6 months of IV antibiotics even if it were fully covered by their insurance company. It is not just an antibiotic deficiency we are encountering; read the book BEYOND ANTIBIOTICS!

It is like the old adage TEACH a man to fish or give him a fish; I prefer the teaching approach. Knowledge of what is really wrong with our health can be empowering but to put everything on one infection or one toxin and ignore leaky gut and food sensitivities, etc I feel  means we provide little long-term meaningful help to patients who deserve a broader understanding of what is really going wrong with their health.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

A Case of Ascending Paralysis: the Signs and Symptoms of Tick Paralysis
Menyoli Malafa, MSII; Veronica Tucci, JD, MS IV; Albert Vincent, PhD; Sajeel Chowdhary, MD
Posted: 03/26/2009; American Academy of Emergency Medicine.
2009;16(1):22, 26, 27 © 2009 American Academy of Emergency Medicine
http://www.medscape.com/viewarticle/589591

Summary
Tick paralysis (TP), a response to the neurotoxic effects of the salivary secretions produced by attached hard ticks (Ixodidae), is a syndrome that mimics a large number of better known neurological disorders. TP is a sporadic, seasonal, rural disorder in which acute ataxia often develops five to six days following a history of walking in grass or low brush, followed by ascending flaccid paralysis. Recognition and timely removal of the tick usually leads to complete resolution of symptoms, whereas continued feeding can lead to respiratory arrest and death. Follow-up includes species determination and patient surveillance for tick-borne infectious disease.

Discussion
TP is a worldwide disease, occurring in Australia, Europe, South Africa and throughout North America. In the United States, most cases occur in the Rocky Mountain states and the Pacific Northwest, including Washington, Montana, Oregon, Idaho, Wyoming, Nevada, Utah, Colorado and the northern parts of Arizona, New Mexico and California. However, cases have also been reported in central, southern and eastern states, including Texas, Oklahoma, Mississippi, Florida, Georgia, North Carolina, South Carolina, Virginia, Washington, D.C., Pennsylvania and New York. In Canada, most cases are encountered in the western part of the country, primarily southern British Columbia.[1,2] More than 60 species of ticks are known to cause paralysis, but only a handful are responsible for most cases. In North America, the disease is associated primarily with six species: Dermacentor andersoni (‘Rocky Mountain wood tick’), D. variabilis (‘American dog tick’), Amblyomma americanum (‘Lone Star tick’), A. maculatum (Gulf Coast tick), Ixodes scapularis (formerly I. dammini, ‘Blacklegged tick’) and I. pacificus (‘Western Black-legged tick’). Peak incidence occurs between April and June when nymphs and mature adults abound in low vegetation and climb upward, questing for their next host by extending their anterior pairs of legs.[1,3,4] Paralysis is a response to a neurotoxin secreted by the salivary glands of the arachnid.[1,5] The biochemistry and pharmacology of the specific paralysis- inducing toxins produced in North American ticks are yet to be fully elucidated, but current evidence points to a mechanism by which the toxins inhibit presynaptic acetylcholine release at the neuromuscular junction.[1,3,6] TP presents more often and more severely in children, suggesting a concentration-dependent relationship between toxin levels and symptom expression.[1,4] Signs and symptoms of TP begin about five to six days after the parasite has attached, when neurotoxin is secreted at its peak levels. These prodromal symptoms include restlessness, irritability, fatigue, nausea, paresthesias and possibly ataxia. Over the next 24-48 hours, the patient develops ascending symmetrical flaccid paralysis and weakness in the lower extremities. Over the course of the next day or two, paralysis and weakness may ascend to involve the trunk, axial and upper limb muscles. Cranial nerves may also become involved in an ascending pattern, resulting in bulbar, facial and/or extraocular paralysis. Patients demonstrate diminished or absent deep tendon and superficial reflexes while, aside from occasional paresthesias, their sensory exam remains normal. Pain and fever are absent. Death ensues following paralysis of the respiratory muscles.[1,5,7,8,9] Atypical presentations reflect variations in the site of tick attachment. There may be ataxia and associated cerebellar deficits without accompanying muscle weakness. The disorder may also present as an isolated facial paralysis without trunk or limb involvement. Another group of atypical presentations is unilateral paralysis and/or weakness, including isolated unilateral facial paralysis.[1,8] Tick paralysis is treated by removal of the tick. Although the site of attachment is most often the head and neck region, the entire body should be scrutinized, including ear canals, nostrils and genitalia. Multiple ticks should be suspected, and all must be removed.[1,4,7,10] Applications of petroleum jelly, nail polish, alcohol, a needle and heat are inappropriate. These measures may result in infection and cause the parasite to salivate or regurgitate more of its bodily fluids.
The tick should be grasped with blunt, angled forceps as close as possible to the skin and to the embedded mouthparts (hypostome). Wearing protective gloves, slowly pull the organism straight outward with a gentle and steady traction, without twisting its body. Do not burst the tick. The hypostome is usually deeply and firmly embedded and should be removed surgically should it come detached. Antiseptic solution is then applied to the wound, and the recovered tick and severed mouthparts may be preserved in 75% ethanol for identification. The patient should be instructed to return in the event of additional illness and educated on protective measures against ticks.
The symptoms of TP, at least those caused by North American species, typically resolve rapidly following removal of all ticks from the patient. Improvement in the condition of the patient subsequent to tick removal is confirmatory for the diagnosis. Species found in some other parts of the world, notably Ixodes holocyclus of Australia, produce a very potent neurotoxin and symptoms may not subside as quickly, even worsening after removal.[5] The prognosis depends on clinical presentation prior to removal. If all ticks were removed prior to the onset of bulbar weakness, the patient often makes a full recovery within the first 24 hours. However, if onset of bulbar symptoms occurs during continued feeding, the likelihood of fatal respiratory paralysis increases to 10%. Therefore, prompt of diagnosis and tick removal are paramount.[1,5,7,8] Because ticks are both vectors and reservoirs for various infectious diseases, it is important to educate the patient about this added risk for possible concurrent illnesses. Table 1 displays the geographical location and infectious diseases associated with North American tick species which are also known to cause TP.[1,8,11,12]

References
1.Cunha BA, editor. Tickborne Infectious Diseases Diagnosis and Management. New York: M. Dekker; 2000.
2.Meier J, White J. Handbook of Clinical Toxicology of Animal Venoms and Poisons. STATE: CRC Press; 1995.
3.CDC. Tick paralysis – Washington. Morbidity and Mortality Weekly Report 1996; 45(16): 325-6.
4.Schmitt N, Bowmer EJ, Gregson JD. Tick paralysis in British Columbia. Can Med Assoc J 1969 Mar 1; 100(9): 417-21.
5.Meriggioli MN, Howard JF, Howard Jr. JF, Harper CM, Harper Jr. CM. Neuromuscular Junction Disorders: Diagnosis and Treatment. STATE: Informa Health Care; 2003.
6.Grattan-Smith PJ, Morris JG, Johnston HM, Yiannikas C, Malik R, Russel R, Ouvrier RA. Clinical and neurophysiological features of tick paralysis. Brain 1997 Nov;120(Pt 11):1975-87.
7.CDC. Tick paralysis – Colorado. Morbidity and Mortality Weekly Report 2006 Sep 1; 55(34): 933-5.
8.Knoop KJ, Stack LB, Storrow AB. Atlas of Emergency Medicine. STATE: McGraw-Hill Professional; 2002.
9.Biller J. Practical Neurology. STATE: Lippincott Williams and Wilkins; 2002.
10.Gammons M, Salam G. Tick removal. Am Fam Physician 2002 Aug 15; 66(4): 646.
11.Winn WC, Kineman EW, Allen SD, Janda WM, Schreckenberger PC,Procop GW, Woods GL. Koneman´s Color Atlas and Textbook of Diagnostic Microbiology. STATE: Lippincott Williams and Wilkins; 2005.
12.Sonenshine DE, Mather TN. Ecological Dynamics of Tick-borne Zoonoses. STATE: Oxford University Press US; 1994.
13.Greenberg BM. Clinical cases in neurology from John Hopkins. Case 2: acute ascending paralysis in a 4-year-old body. MedGenMed 2003 Apr 9; 5(2): 36.

Excerpt:

A 
number of studies previously published, and new information presented 
here, suggest that EA infections may be an underlying, undiagnosed cause 
for these and other immune system diseases. This hypothesis, long 
overlooked, has never been subjected to adequate, rigorous study 
sufficient to prove or disprove its truth. If so, patients may be 
treated with antibiotics, and marrow transplant manipulations already 
used in treatment of diseases such as lupus and leukemia may become more 
effective.

Dr. Gordon’s Comments:

WSJ says the new epidemic is gluten sensitivity and it is different than celiac disease but is now being blamed for multiple common symptoms. It is not easy to live gluten free since it seems to be everywhere. See The Gluten Connection by Shari Lieberman and others on Amazon.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com

Link: http://drhyman.com/gluten-what-you-dont-know-might-kill-you-11/?utm_source=Publicaster&utm_medium=email&utm_campaign=drhyman%20newsletter%20issue%20#17&utm_term=Get+the+story

Excerpt:

Gluten: What You Don’t Know Might Kill You
Dr Mark Hyman

SOMETHING YOU’RE EATING may be killing you, and you probably don’t even know it! If you eat cheeseburgers or French fries all the time or drink six sodas a day, you likely know you are shortening your life. But eating a nice dark, crunchy slice of whole wheat bread–how could that be bad for you? Well, bread contains gluten, a protein found in wheat, barley, rye, spelt, kamut, and oats. It is hidden in pizza, pasta, bread, wraps, rolls, and most processed foods. Clearly, gluten is a staple of the American diet. What most people don’t know is that gluten can cause serious health complications for many. You may be at risk even if you don’t have full blown celiac disease. I want to reveal the truth about gluten, explain the dangers, and provide you with a simple system that will help you determine whether or not gluten is a problem for you.

 Link: http://online.wsj.com/article/SB40001424052748704893604576200393522456636.html?mod=djempersonal 

Excerpt: Wall Street Journal
•HEALTH JOURNAL 
•MARCH 16, 2011 
Gluten sensitive? Here’s why 
By Melinda Beck

Lisa Rayburn felt dizzy, bloated and exhausted. Wynn Avocette suffered migraines and body aches. Stephanie Meade’s 4-year-old daughter had constipation and threw temper tantrums. 

All three tested negative for celiac disease, a severe intolerance to gluten, a protein found in wheat and other grains. 

But after their doctors ruled out other causes, all three adults did their own research and cut gluten—and saw the symptoms subside. A new study in the journal BMC Medicine may shed some light on why. It shows gluten can set off a distinct reaction in the intestines and the immune system, even in people who don’t have celiac disease.

Excerpt:

Using cryo-electron tomography, we observed closely associated Borrelia cells. Some of these showed a single outer membrane surrounding two longitudinally arranged cytoplasmic cylinders. We also observed fusion of two cytoplasmic cylinders and differences in the surface layer density of fused spirochetes. These processes could play a role in the interaction of Borrelia species with the host’s immune system.

Excerpt:

Some Lyme disease patients report debilitating chronic symptoms of pain, fatigue, and cognitive deficits despite recommended courses of antibiotic treatment. The mechanisms responsible for these symptoms, collectively referred to as post-Lyme disease syndrome (PLS) or chronic Lyme disease, remain unclear. We investigated the presence of immune system abnormalities in PLS by assessing the levels of antibodies to neural proteins in patients and controls. Serum samples from PLS patients, post-Lyme disease healthy individuals, patients with systemic lupus erythematosus, and normal healthy individuals were analyzed for anti-neural antibodies by immunoblotting and immunohistochemistry. Anti-neural antibody reactivity was found to be significantly higher in the PLS group than in the post-Lyme healthy (p<0.01) and normal healthy (p<0.01) groups. The observed heightened antibody reactivity in PLS patients could not be attributed solely to the presence of cross-reactive anti-borrelia antibodies, as the borrelial seronegative patients also exhibited elevated anti-neural antibody levels. Immunohistochemical analysis of PLS serum antibody activity demonstrated binding to cells in the central and peripheral nervous systems. The results provide evidence for the existence of a differential immune system response in PLS, offering new clues about the etiopathogenesis of the disease that may prove useful in devising more effective treatment strategies. Copyright 2010 Elsevier Inc. All rights reserved.

Results

We demonstrate that B. garinii OspA serotype 4 (ST4) PBi resist complement-mediated killing by binding of FHL-1. To identify the primary ligands of FHL-1 four CspA orthologs from B. garinii ST4 PBi were cloned and tested for binding to human CFH and FHL-1. Orthologs BGA66 and BGA71 were found to be able to bind both complement regulators but with different intensities. In addition, all CspA orthologs were tested for binding to mammalian and avian CFH. Distinct orthologs were able to bind to CFH of different animal origins.

Conclusions

B. garinii ST4 PBi is able to evade complement killing and it can bind FHL-1 to membrane expressed proteins. Recombinant proteins BGA66 can bind FHL-1 and human CFH, while BGA71 can bind only FHL-1. All recombinant CspA orthologs from B. garinii ST4 PBi can bind CFH from different animal origins. This partly explains the wide variety of animals that can be infected by B. garinii

Excerpt:

Methods

Human leukemic cultured LAD2 mast cells and normal human umbilical cord blood-derived cultured mast cells (hCBMCs) were stimulated by HgCl2 (0.1-10 microM) for either 10 min for beta-hexosaminidase release or 24 h for measuring vascular endothelial growth factor (VEGF) and IL-6 release by ELISA.

Results

HgCl2 induced a 2-fold increase in beta-hexosaminidase release, and also significant VEGF release at 0.1 and 1 microM (311+/-32 pg/10*6 cells and 443+/-143 pg/10*6 cells, respectively) from LAD2 mast cells compared to control cells (227+/-17 pg/10*6 cells, n=5, p<0.05). Addition of HgCl2 (0.1 microM) to the proinflammatory neuropeptide substance P (SP, 0.1 microM) had synergestic action in inducing VEGF from LAD2 mast cells. HgCl2 also stimulated significant VEGF release (360 +/- 100 pg/10*6 cells at 1 microM, n=5, p<0.05) from hCBMCs compared to control cells (182 +/-57 pg/10*6 cells), and IL-6 release (466+/-57 pg/10*6 cells at 0.1 microM) compared to untreated cells (13+/-25 pg/10*6 cells, n=5, p<0.05). Addition of HgCl2 (0.1 microM) to SP (5 microM) further increased IL-6 release.

Conclusions

HgCl2 stimulates VEGF and IL-6 release from human mast cells. This phenomenon could disrupt the blood-brain-barrier and permit brain inflammation. As a result, the findings of the present study provide a biological mechanism for how low levels of mercury may contribute to ASD pathogenesis.

  Lethal and toxic levels of hydrogen sulfide, benzene, and methalene chloride are floating in the air over the oil spill. There’s a very high probability that residents exposed to the air surrounding the spill will suffer a direct hit to their health status such as debilitating diseases or various birth deformities and cancer as a long-term result. But first what these people will see is flu-like symptoms, which, like in the flu, are symptoms of intolerable amounts of foreign toxins, chemicals and heavy metals in the tissues dumping into the bloodstream.
 
     Even a small amount of benzene exposure can cause temporary nervous system disorders, immune system depression and anemia. Short-term affects include skin, eye, and respiratory tract irritation, headache, stomach irritation, drowsiness and dizziness. High levels of exposure can result in a rapid heart rate, excessive bleeding, tremors, vomiting, unconsciousness and death. Benzene can cause harmful effects on bone marrow and a decrease in red blood cells leading to myelofibrosis and myelodysplastic syndrome.
 
     That’s how it starts. Chemical exposure symptoms feel like a flu. Professor I.M. Trakhtenberg of Russia gives us a big hint when he says, “Chronic mercury exposure is also a threat to our health and makes us especially vulnerable to flu infections. It has been shown that “prolongedexposure of mammals (white mice) to low mercury concentrations (0.008 – 0.02mg/m3) leads to a significant increase in the susceptibility of mice topathological influenza virus strains.” For contemporary medicine to respond in an appropriate and humane way to the oil disaster it will have to leap out of the quagmire of its present paradigm an into one that understands the ‘terrain’ of human physiology and how that terrain is being overrun by chemical toxicity and heavy metals. WE DO NOT NEED TO BE ATTACKED BY AN INFLUENZA VIRUS STRAIN TO GET THE FLU. When we are attacked with nasty chemicals we are as likely to get the flu as when we are run over by viruses, which are more potent at driving health officials mad as at causing pandemics.
 
     “Blood elements such as WBCs, RBCs, hemoglobin, and bone marrow are adversely affected. With tissue proteins there is alteration of biological properties and protein synthesis. Enzyme; hormone; and endocrine functions of pituitary, adrenal, thyroid, ovaries, and testes are altered. There are pathological effects on the heart, liver, immune system, central nervous system, lungs, kidneys, and spleen.” continues Dr. Trakhtenberg.
 
     Thiol poisons react with SH groups of proteins, which leads to lowering the activity of various enzymes containing these proteins. This produces a series of disruptionsin the functional activity of many organs and tissues and this is the mechanism and pathological pathway of poisons that run us right into the ground. A toxic storm is gathering in the Gulf of Mexico and it contains devastating chemicals that can and will poison and destroy proteins with sulfur bonds.
 
Associated Illnesses
 
     According to the U.S. Department of Veterans Affairs, between 175,000 and 210,000 – or about 25 percent – of the living veterans of the 1991 Gulf War are currently afflicted by a debilitating, chronic, multi-symptom, multi-system disease commonly known as Gulf War Illness or Gulf War Syndrome. The Environmental Illness Resource , (http://imva.us1.list-manage.com/track/click?u=25b08cc8b5ebaf472984d04d0&id=f7a015aaa4&e=a053e43583) tells us that more than 110,000 cases had been reported by 1999, according to official government sources. There is even a report relating to military personnel in Kansas developing flu-like symptoms and chemical sensitivities after handling archived documents returned from the Gulf. In the UK, veterans of the 2003 conflict began reporting symptoms identical to those reported by the first war shortly after they returned from duty.
 
     The symptoms reported by veterans include:
 
Fatigue
Persistent Headaches
Muscle Aches/Pains
Neurological Symptoms, e.g. tingling and numbness in limbs
Cognitive Dysfunction – short-term memory loss, poor concentration, inability to take in information
Mood and Sleep Disturbances – Depression, Anxiety, Insomnia
Dermatological Symptoms – Skin Rashes, Unusual Hair Loss
Respiratory Symptoms – Persistent Coughing, Bronchitis, Asthma
Chemical Sensitivities
Gastrointestinal Symptoms – Diarrhea, Constipation, Nausea, Bloating
Cardiovascular Symptoms
Menstrual Symptoms
 
     These symptoms are similar to those attributed to chronic fatigue syndrome, multiple chemical sensitivities and other environmental illnesses. This similarity hasn’t gone unnoticed, which is why many people, including healthcare professionals and researchers, are coming to the conclusion that all these illnesses share common causes and etiologies. Gulf War vets have developed ALS, or Lou Gehrig’s disease, at twice the rate of vets who did not serve in the Gulf War. Some veterans returned seemingly well, yet developed severe illnesses months or years later. The lag time between cause and effect makes understanding these illnesses more difficult.
 
     Coalition troops were constantly exposed to chemicals (and vaccines) whose use is considered safe by people and organizations that do not know a safe substance from a dangerous one. The retreating Iraqi army ignited approximately 600 oil wells in February 1991, which burned for about nine months. These fires produced massive amounts of thick smoke that sometimes drifted to ground level causing increased exposure to ground troops. When this occurred the air pollution was far greater than would be experienced in the average traffic congested western city.
 
     Questionnaires filled in by US troops indicated higher rates of eye and upper respiratory tract irritation, shortness of breath, cough, rashes, and fatigue than unexposed troops. The smoke from oil well fires contained a cocktail of chemicals, notably benzene, hydrogen sulfide and sulfur dioxide as well as quantities of particulate matter.
 
Read The Full Article
Mark Sircus Ac., OMD
Director International Medical Veritas Association
http://publications.imva.info
http://blog.imva.info
 

Were you on aspartame before you started using Splenda?  Before
Splenda we thought never could anyone ever create another poison like
aspartame that is so toxic it destroys the brain, the central nervous
system, the optic nerve and the immune system, and ravages every
organ in the body.  From taking the case histories for 19 years I
can’t imagine anything more poisonous.  Even with testing on
aspartame the FDA wanted them indicted for fraud because there was no
way to show safety and its a carcinogen.  But when Splenda came along
consumers were aghast they had made something else that poisons the
system.  To make matters worse, Dr. James Bowen says that
if  consumers go from aspartame to Splenda they will maintain the
reactions from aspartame and pick up those from Splenda.

Using Splenda is like drinking bleach and today reactions and severe
ones are showing up on Splenda.    You might want to read
this:  http://www.holisticmed.com/splenda/splenda-adverse.txt and
http://articles.mercola.com/sites/articles/archive/2004/03/31/splenda-reaction.aspx
In today’s society with companies poisoning our food supply you have
to become a label reader.  The FDA doesn’t care about the food supply
being safe.  So you must be responsible for what you put in your mouth.

One thing we know is that Splenda interacts.  The company admitted to
Marianne Lamar that  Splenda does cause headaches and seizures.  That
means you are using a deadly drug, its not inert.  This came directly
from the company itself although there are many reactions to Splenda
from rashes to cardiac problems.  As to joint pain, yes it has been
reported with Splenda.  Read this report:
http://splendasickness.blogspot.com/2006/03/joint-pain-or-swelling.html

Also, Citizens for Health and James Turner, DC attorney filed a
petition to ban Splenda and they haven’t received an answer although
there is a time limit on the FDA answering citizens petitions which
is 180 days.  They haven’t answered my petition for ban of aspartame
in almost 7 years.  So you know the FDA is serving above the law and
giving their loyalty to Big Pharma and the chemical industry.  This
is something Congress needs to deal with.  In fact, I’ve petitioned
the FDA to ban aspartame based on an imminent health hazard and they
only have a week or ten days to answer that.  That was over two years
ago.   So you have an FDA way out of control who is ignoring
consumers  and scientists.  Dr. Stoller also petitioned the FDA to
ban aspartame and its not been answered.  In fact, twelve
toxicologists have written the FDA to ban aspartame, and I doubt that
they have heard either.

The email for Citizens for Health is info@citizens.org and I’m
sending a copy of this to James Turner.  On the Aspartame Resource
Guide I sent you the physicians have detoxification help.  Then you
could have prolotherapy which cures chronic pain. www.caringmedical.com

Here are some symptoms reported on Splenda:

Sucralose aka Splenda Can Cause Pain & Tension

Sucralose Can Cause These Symptoms

Abdominal pain, achiness, back pain, chest tightness, dizziness, eye
pain, fibromyalgia, headache, joint pain, leg cramps, loss of
equilibrium, migraine, numbness & tingling of hands & feet, shooting
pains in extremities, swallowing pain, tingling, unsteady gait, weakness, more.

You can subscribe to the Aspartame Information List on
www.mpwhi.com   (scroll down to banners) as we also take Splenda
reactions and its discussed.

Just Like Sugar (www.justlikesugarinc.com) is safe and can be gotten
in most Whole Foods.

I would definitely fill out the MedWatch form on the FDA to have your
complaints on record. Do send me a copy. Remember that Sucralose has
already been found in drinking
water.
http://www.scientificamerican.com/blog/60-second-science/post.cfm?id=that-splenda-youre-drinking-will-be-2009-03-09
Read about detoxification using distilled water.

All my best,
Betty

Dr. Betty Martini, D.Hum, Founder
Mission Possible International
9270 River Club Parkway
Duluth, Georgia 30097
770 242-2599
www.mpwhi.com, www.dorway.com, www.wnho.net
Aspartame Toxicity Center, www.holisticmed.com/aspartame