link: http://www.medicationsense.com/fluoroquinolone.html 

Excerpt:

Levaquin and Cipro Reactions

In 2001, Dr. Jay S. Cohen published a ground-breaking article* on the severe and often disabling reactions some people sustained while taking Levaquin, Cipro, or another FQ antibiotic. Dr. Cohen says, “It is difficult to describe the severity of these reactions. They are devastating. Many of the people in my study were healthy before their reactions. Some were high intensity athletes. Suddenly they were disabled, in terrible pain, unable to work, walk, or sleep.” 
The 45 subjects in Dr. Cohen’s study reported the following side effects*.

Peripheral Nervous System

: Tingling, numbness, prickling, burning pain, pins/needles sensation, electrical or shooting pain, skin crawling, sensation, hyperesthesia, hypoesthesia, allodynia (sensitivity to touch), numbness, weakness, twitching, tremors, spasms.

Central Nervous System:

 

Dizziness, malaise, weakness, impaired coordination, nightmares, insomnia, headaches, agitation, anxiety, panic attacks, disorientation, impaired concentration or memory, confusion, depersonalization, hallucinations, psychoses.

  Lethal and toxic levels of hydrogen sulfide, benzene, and methalene chloride are floating in the air over the oil spill. There’s a very high probability that residents exposed to the air surrounding the spill will suffer a direct hit to their health status such as debilitating diseases or various birth deformities and cancer as a long-term result. But first what these people will see is flu-like symptoms, which, like in the flu, are symptoms of intolerable amounts of foreign toxins, chemicals and heavy metals in the tissues dumping into the bloodstream.
 
     Even a small amount of benzene exposure can cause temporary nervous system disorders, immune system depression and anemia. Short-term affects include skin, eye, and respiratory tract irritation, headache, stomach irritation, drowsiness and dizziness. High levels of exposure can result in a rapid heart rate, excessive bleeding, tremors, vomiting, unconsciousness and death. Benzene can cause harmful effects on bone marrow and a decrease in red blood cells leading to myelofibrosis and myelodysplastic syndrome.
 
     That’s how it starts. Chemical exposure symptoms feel like a flu. Professor I.M. Trakhtenberg of Russia gives us a big hint when he says, “Chronic mercury exposure is also a threat to our health and makes us especially vulnerable to flu infections. It has been shown that “prolongedexposure of mammals (white mice) to low mercury concentrations (0.008 – 0.02mg/m3) leads to a significant increase in the susceptibility of mice topathological influenza virus strains.” For contemporary medicine to respond in an appropriate and humane way to the oil disaster it will have to leap out of the quagmire of its present paradigm an into one that understands the ‘terrain’ of human physiology and how that terrain is being overrun by chemical toxicity and heavy metals. WE DO NOT NEED TO BE ATTACKED BY AN INFLUENZA VIRUS STRAIN TO GET THE FLU. When we are attacked with nasty chemicals we are as likely to get the flu as when we are run over by viruses, which are more potent at driving health officials mad as at causing pandemics.
 
     “Blood elements such as WBCs, RBCs, hemoglobin, and bone marrow are adversely affected. With tissue proteins there is alteration of biological properties and protein synthesis. Enzyme; hormone; and endocrine functions of pituitary, adrenal, thyroid, ovaries, and testes are altered. There are pathological effects on the heart, liver, immune system, central nervous system, lungs, kidneys, and spleen.” continues Dr. Trakhtenberg.
 
     Thiol poisons react with SH groups of proteins, which leads to lowering the activity of various enzymes containing these proteins. This produces a series of disruptionsin the functional activity of many organs and tissues and this is the mechanism and pathological pathway of poisons that run us right into the ground. A toxic storm is gathering in the Gulf of Mexico and it contains devastating chemicals that can and will poison and destroy proteins with sulfur bonds.
 
Associated Illnesses
 
     According to the U.S. Department of Veterans Affairs, between 175,000 and 210,000 – or about 25 percent – of the living veterans of the 1991 Gulf War are currently afflicted by a debilitating, chronic, multi-symptom, multi-system disease commonly known as Gulf War Illness or Gulf War Syndrome. The Environmental Illness Resource , (http://imva.us1.list-manage.com/track/click?u=25b08cc8b5ebaf472984d04d0&id=f7a015aaa4&e=a053e43583) tells us that more than 110,000 cases had been reported by 1999, according to official government sources. There is even a report relating to military personnel in Kansas developing flu-like symptoms and chemical sensitivities after handling archived documents returned from the Gulf. In the UK, veterans of the 2003 conflict began reporting symptoms identical to those reported by the first war shortly after they returned from duty.
 
     The symptoms reported by veterans include:
 
Fatigue
Persistent Headaches
Muscle Aches/Pains
Neurological Symptoms, e.g. tingling and numbness in limbs
Cognitive Dysfunction – short-term memory loss, poor concentration, inability to take in information
Mood and Sleep Disturbances – Depression, Anxiety, Insomnia
Dermatological Symptoms – Skin Rashes, Unusual Hair Loss
Respiratory Symptoms – Persistent Coughing, Bronchitis, Asthma
Chemical Sensitivities
Gastrointestinal Symptoms – Diarrhea, Constipation, Nausea, Bloating
Cardiovascular Symptoms
Menstrual Symptoms
 
     These symptoms are similar to those attributed to chronic fatigue syndrome, multiple chemical sensitivities and other environmental illnesses. This similarity hasn’t gone unnoticed, which is why many people, including healthcare professionals and researchers, are coming to the conclusion that all these illnesses share common causes and etiologies. Gulf War vets have developed ALS, or Lou Gehrig’s disease, at twice the rate of vets who did not serve in the Gulf War. Some veterans returned seemingly well, yet developed severe illnesses months or years later. The lag time between cause and effect makes understanding these illnesses more difficult.
 
     Coalition troops were constantly exposed to chemicals (and vaccines) whose use is considered safe by people and organizations that do not know a safe substance from a dangerous one. The retreating Iraqi army ignited approximately 600 oil wells in February 1991, which burned for about nine months. These fires produced massive amounts of thick smoke that sometimes drifted to ground level causing increased exposure to ground troops. When this occurred the air pollution was far greater than would be experienced in the average traffic congested western city.
 
     Questionnaires filled in by US troops indicated higher rates of eye and upper respiratory tract irritation, shortness of breath, cough, rashes, and fatigue than unexposed troops. The smoke from oil well fires contained a cocktail of chemicals, notably benzene, hydrogen sulfide and sulfur dioxide as well as quantities of particulate matter.
 
Read The Full Article
Mark Sircus Ac., OMD
Director International Medical Veritas Association
http://publications.imva.info
http://blog.imva.info
 

 HRT Doubles Recurrence Risk in Breast Cancer Survivors

LONDON, March 25 — Breast cancer survivors treated with hormone replacement therapy had a more than two-fold increased risk of recurrence or a contralateral malignancy, according to long-term follow-up data from a randomized clinical trial.

•Note that this is one of the few randomized, controlled studies that have examined the risk of breast cancer recurrence associated with HRT.
Those randomized to HRT had five-year breast cancer rates of 22.2% compared with 8% in women who received best patient care for menopausal symptoms without hormone therapy, Lars Holmberg, M.D., Ph.D., of King’s College London, and colleagues, reported in the April 2 issue of the Journal of the National Cancer Institute.

“The results of the HABITS [Hormonal Replacement after Breast Cancer — Is It Safe?] trial indicate a substantial risk a new breast cancer event among breast cancer survivors using hormone therapy,” the authors concluded.

“Our results further suggest that hormone therapy not only induces and promotes breast cancer but may also stimulate the growth of tumor microdeposits in breast cancer survivors,” they added.

Despite the statistically significant impact of hormone therapy on breast cancer risk, the authors said more data from randomized studies are needed to define the risk and to clear up inconsistencies in prior studies.

However, Kathleen Pritchard, M.D., a breast cancer specialist at Sunnybrook Odette Cancer Center in Toronto, said in an accompanying editorial that the study “suggests quite definitively that there is a statistically significantly increased risk of recurrence in women given HRT following diagnosis of breast cancer.”

Persistent questions about the potential risks and benefits of HRT in breast cancer survivors provided impetus for several observational studies and analyses of case series. More recently, data from the Women’s Health Initiative and the Million Women Study provided additional compelling evidence of an increased risk of breast cancer among HRT users, the King’s College authors said.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com

From: John Cannell, M.D.
The Vitamin D Newsletter
January 30, 2010.

Dear Dr. Cannell:
At age 2.5 years, between December 2007 and January 2008, my son experienced a fairly dramatic onset of symptoms that led to his diagnosis of autism. His symptoms (many of which we did not even know the terminology for at the time they first occurred) included:
–The inability to sleep at night, we would put him to bed at 8:00 or 8:30 p.m. following his normal bedtime routine
–Development of anxiety and refusal to leave the house even to do preferred activities
–Obsessive-repetitive questions and monologuing/run-on speech
–Sensory issues (refusal to wear jeans or any fabrics other than fleece, screaming hysterically at bath time, complaining and covering eyes in sunlight, covering ears for everyday noises that had not bothered him before (toilets flushing, pulling pots and pans from cupboards, etc.)
–Toe-walking
–Flapping and self-stimulating behaviors (repeatedly tapping his cheeks and eyes with all ten fingers, continually twisting up his fingers in pretzel-like configurations, holding objects in his peripheral range of vision and straining to see them from the corner of his eyes)
–Development of an unusual pattern of stuttering/vocal tic at the end of words,he would repeat the last sound/syllable,”I don’t want to go to the store-or-or-or-or-or-or. It won’t be fun-n-n-n-n-n-n-n.” He would make sounds even in his sleep “n-n-n-n-n-n” or “s-s-s-s-s-s-s”
–Loss of muscle tone (stopped walking up and down stairs and began crawling/sliding instead, decline in balance and motor skills)
–loss of handedness (began switching left to right hand, after seeming predominantly left-handed)
–Marked increase in hyperactivity
–Frequent spacing out/unresponsive episodes

Our son and his twin sister were born at 36 weeks, 5 days on March 17, 2005 after four months of bed-rest. As early as their 8 week appointment, I mentioned to our pediatrician that we had concerns about our son’s eye contact and social responsiveness (in comparison to his sister). I felt that I was having more difficulty bonding with him. We were told “don’t worry, but don’t wait” and were referred to our state’s Early On intervention program. At the end of June a physical therapist and speech pathologist from our intermediate school district came to our home to evaluate our then 3 month old son and told me that he was doing just fine and that I was worrying too much. I agreed that by the time they saw him he had begun smiling and making better eye contact.
We didn’t worry again about our son until fall 2006. He had walked just before his first birthday, but by 18 months+ he still seemed clumsy and prone to falling compared to his sister. We took him back to the intermediate school district for evaluation and were told that all of his development seemed to be in the normal range and that we shouldn’t worry. We were advised that we could take him to music and gym classes to work on his coordination and told that we could pay for private physical therapy if we elected. We followed all of the recommendations.

For a year, we didn’t notice any other changes until the sudden onset of symptoms listed above when he was 2.5 years. With the sudden onset of symptoms above, we took our son to see a number of specialists during the winter of 2008 including a neurologist (who diagnosed him with Asperger Syndrome), a psychologist (who diagnosed with autism), and a second psychologist who specialized in the treatment of autism (who diagnosed him with Pervasive Developmental Disorder Not-Otherwise-Specified). All three diagnoses are on the autism spectrum. He also began seeing an occupational therapist, a speech therapist, a behavioral specialist, and a DAN! (Defeat Autism Now!) doctor for dietary interventions. We saw a dramatic improvement by April/May of that year. Nearly all the symptoms on the list above had resolved. We assumed the improvements were due to diet but he started to go into the sun around that time. Our son slept well and spent many peaceful, happy and anxiety-free months during the spring and summer after turning three.

In mid-November 2008, I sent the following e-mail to the DAN doctor who had been helping us with our son.

“You saw our son Jonathan Switzer a few times regarding his autism diagnosis and diet issues, etc. He had a regressive period last winter from about December through April when his autism was diagnosed, then did pretty well all summer. Nursery school started off okay, too, but now he seems to be having another regression. 

Main symptoms:
–Great difficulty getting to sleep (fidgets for 2 plus hours most nights while he had been falling asleep easily for several months prior to that)
–Marked increase in anxiety (again refusing to leave the house even to do things he loves, frequently shaking/clenching and telling us “I’m scared)
–Onset of OCD-like behaviors (afraid to get hands dirty, get extremely upset if he gets even tiny drips of water on himself)
–Increase in self-stimulatory behaviors (flapping, fidgeting, noise-making)
–Frequent crying jags and telling us he’s just giving up on everything

We have had other parents tell us that their kids on the spectrum have a worsening of symptoms during the winter months and we feel like we are observing this same pattern. We’ve done some reading about light therapy for depression/anxiety and to help correct disturbed sleep patterns and would like to give it a try for Jonathan.

Wondering if you have ever prescribed a light therapy box for pediatric patients before. Our insurance told us they will cover it with a diagnosis of Seasonal Affective Disorder, but I don’t even know if that is something that can be diagnosed in children. Guess we’re willing to try anything at this point. Do you know much about this type of therapy?”
Neither the DAN Doctor nor our pediatrician would write a prescription for a therapy light, so we purchased one on our own and found it made no discernible impact on his symptoms.
By December, our son’s symptoms had worsened further and we decided to put him in a very expensive and intensive autism treatment program through our local hospital. He made slow progress during his participation in the program from January through April. He was also involved in speech and occupational therapy during the winter months. At his IEPC meeting at school in March, we were encouraged to put him in the district’s program for children with developmental delays. We instead elected to register him for regular pre-school for the following year.

During that winter, I was crying to some friends about my son and describing his seemingly seasonal pattern of symptoms. We had just seen a second neurologist searching for help, and I was extremely frustrated when, after listening to my son’s symptoms and history, he told me bluntly, “There is nothing seasonal about autism,” then suggested that we put our son on an anti-depressant. We refused the medication. One of the friends I was crying to is a research librarian and the other is a medical researcher. After our conversation, they located and e-mailed me a few journal articles they thought might help, one of the articles was by Dr. Cannell and discussed his vitamin D theory of autism. Reading the article was one of those “Aha!” moments and I felt hopeful that Dr. Cannell was on to something.

By June our son was released from both speech therapy and occupational therapy and we were told that he no longer showed any delays for his age. When he had begun occupational therapy in January, the OT had been astonished at our son’s lack of muscle tone. She recommended that he also receive Physical Therapy services, so we went on a long waiting list. Our initial OT was in a car accident, and in May we were transferred to a new OT. When the new OT first saw our son, she said could not believe he was the same child described in the notes. By May the low muscle tone, hyperactivity and distractibility noted in his file, were no longer evident. His turn came up for physical therapy and we were told he no longer needed it.

Our son has always spent a lot of time outdoors in the summer, without sunblock. He had a happy and relaxing summer. As fall/back-to-school approached, I began to fear the onset of another regression and again read the article by Dr. Cannell my friend had sent. I visited his website and decided we would try a vitamin D supplement. Our pediatrician did not encourage any dose higher than 400 i.u. (that found in a typical multivitamin) but did write a script to have his 25-hydroxy level tested. In August his level was 37, so we started him on 5,000 iu daily and had his level retested on October 21st. By October his level was 96. The pediatrician was concerned that this was too high and told us he should not have more than 400 iu per day.

Knowing that Nov-March are typically his worst months, we reduced the dosage down only to 3,000 iu from October through mid-December. At an appointment in December our son was doing wonderfully (none of his usual fall/winter symptoms yet evident) and the pediatrician told us 3,000 iu was too much and that we should be giving no more than 400 iu. In mid-December we reduced the dose to 1,500 iu. By the beginning of January we noted a marked loss of eye contact. We also noted that our son was again interchanging his right hand for writing and eating (after using his left hand exclusively for 8+ months). We increased his vitamin D level to 4,000 iu daily in early January. On January 11 we had his 25-Hydroxy level checked on January 11 and found that it was 89. By the end of January, we and his grandparents noted improvement in his eye contact.

In January 2010 we attended his preschool conferences. The teacher had marked cards with the following code (1=age appropriate, 2=developing, 3=area of concern). Our son received 1s in all areas with the exception of hopping on one foot and balance beam where he received 2s. We were told that he is on par with or ahead of his peers in all areas (academic, fine motor, etc.), and that his teacher had noted no unusual symptoms or concerns.

During the fall/winter 2009-2010 our son has been free from nearly all of the most troubling symptoms that plagued him the previous two winters. The following example may demonstrate the improvement in his daily life since last winter.

One of our son’s low points was a Christmas party we attended in December 2008. Before leaving the house to attend the party our son screamed and yelled about having to take a bath and because we would not let him wear sweatpants to the party. He then begged us not to make him leave the house. During the 40 minute trip to the party our son asked us repetitive questions and talked incessantly. Upon arriving at the party, he immediately walked into an unoccupied room adjacent to the room where the party was occurring, and put his face into the corner. Despite much coaxing by my husband and me, he refused to come out of the corner.

After approximately 45 minutes of standing in the corner we managed to get him out through the promise of some food rewards. He proceeded to walk around and around the perimeter of the living room where all of the other kids were playing. He rubbed himself along the walls and covered his ears as he walked. He finally settled into playing alone in a corner of the room. All of the kids at the party participated in a book exchange. Our son refused to come to the area where the other kids were gathered. We coaxed him over only to have him throw the book he received and refuse to thank the parent who had purchased it for him. He spent much of the evening in time-outs for that and other inappropriate behavior.

In June of 2008, after playing in the sun for several months, we met for a picnic with the same group of friends at a local park. Our son ran up to the other children and joined right in playing bulldozers in the sand with them. He behaved and interacted in a completely appropriate and typical way during the picnic which lasted several hours.

This year (2009) we attended the same Christmas party at the same house. Our son got ready and left for the party without anxiety or incident. He chatted normally during the drive to the party. He walked into the house, said, “Hey, check out my new train,” to some of the kids already playing and settled in to playing happily with the other kids. During the book exchange, he received a book, smiled and gave a big hug to the person who gave it to him.

In December of 2008, I took a leave from my job so I could get my son to the intensive behavioral treatment program he was in and to all of his other therapy appointments. I dedicated 40-60 hours per week to my son’s various appointments and home therapy program.

This winter (January 2010), a former colleague asked me what Jonathan’s current therapy program consists of. I told her I spend about 30 seconds each day opening the jar of vitamins and giving him his chewable vitamin D. In my opinion, the 3 minutes or so I spend each week giving him his vitamin D have been much more effective, and much less expensive, than any other treatment we have pursued. 
Thank you.
Jeannette, Wisconsin

Dear Jeanette:
You’re welcome. Several things need comment. First, the symptoms are typical of autism. Second, the seasonality of symptoms suggest a vitamin D deficient disease. Third, the treatment in the spring of 2008 seemed effective but, in hindsight, it was simply due to spring sun exposure. Fourth, as you may now know, light boxes for seasonal affective disorder make no vitamin D. Fifth, your pediatrician knows little about Vitamin D other than what committees tell him; your decision to ignore his advice probably saved your son’s brain from further injury, as autism is a progressive inflammatory destruction of brain tissue. Sixth, the fact that you needed bed rest and gave birth prematurely suggests you were Vitamin D deficient during your pregnancy.

Seventh, his twin sister has never had autism, despite the same intrauterine environment. This is consistent with my theory, that autism is caused from a quantitative, not qualitative, variation is one of the enzymes that metabolize Vitamin D. That is, there are no structural differences in these enzymes in autism, only a genetically determined difference in the amount present. These enzymes are responsive to estrogen; estrogen protects the brain from being damaged by low Vitamin D, probably by increasing the amount of activated Vitamin D present, explaining why boys are four times more likely to have the disease.
The report that your son deteriorated when his dose was reduced from 3,000 to 1,500 IU suggests autistic children need adult doses of Vitamin D. When you reduced the dose from 3,000 to 1,500 IU/day he worsened although his level on 1,500 IU/day was probably still greater than 50 ng/ml. This makes me think that dosage needs to be stable and suggests that Professor Reinhold Vieth’s theory of a detrimental seasonal resetting of the intercellular metabolism of Vitamin D may even be true at levels above 50 ng/ml, where the body is storing the parent compound, cholecalciferol, in muscle and fat.

His current dose of 4,000 IU per day is perfectly safe and will give him a level of 80-100 ng/ml, inside the reference ranges of American laboratories. Toxicity (asymptomatic high blood calcium) begins somewhere above 200 ng/ml. Generally speaking, autistic children should take 2,000 IU per every 25 pounds of body weight for six weeks, then have a 25(OH)D blood test and adjust the dosage to get into the high end of the reference range, 80-100 ng/ml.

Although I first published the Vitamin D theory of autism theory 3 years ago, few autistic children are currently treated for their Vitamin D deficiency. This is due to several reasons. One, those who think, correctly, that autism is a genetic disease, stop thinking after that, reasoning that genetic diseases are untreatable. Such thinkers do not understand epigenetics (upon the genome). Vitamin D is probably the heart of epigenetics, as nothing works upon the genome like vitamin D.

Secondly, the “all autism is caused from vaccinations” crowd cannot accept the Vitamin D possibility as it threatens their core beliefs. They simply cannot change their minds.
Finally, as you now know, organized medicine would say you should stop the vitamin D and watch your son deteriorate, which is why slavery to evidence based medicine is fine for scientists and unethical for practitioners.

John Cannell, MD
Executive Director
Vitamin D Council

Toxic Effects of Excess Mercury

Mercury is considered to be the most toxic non-radioactive Heavy Metal.

Cardiovascular System
Excessive ingestion of Mercury can cause Angina.

Ears/Hearing
Mercury toxicity can be an underlying cause of Deafness. 
Mercury toxicity can be an underlying cause of Tinnitus. 

Excretory System
Mercury accumulates in and damages the Kidneys. 

Immune System
Mercury toxicity can be an underlying cause of Autoimmune Diseases.
Mercury weakens the Immune System by interfering with the balance of Helper T-Cells subpopulations:
Mercury inhibits the activity of TH1 Helper T-Cells. 
Mercury stimulates the overactivity activity of TH2 Helper T-Cells. 
Excessive exposure to Mercury causes Hodgkin’s Disease (a form of Lymphatic Cancer).

Metabolism
Exposure to Mercury (or its vapors) can cause Fatigue. 
Mercury stimulates the production of Free Radicals. 
Mercury accumulates in and damages the Liver.

Musculoskeletal System
Mercury increases the risk of Arthritis.
Exposure to Mercury can cause Muscle Tension. 
Exposure to Mercury can cause Muscle Weakness. 

Nervous System
Exposure to Mercury causes impairment in Abstract Reasoning ability. 
Exposure to Mercury can cause Aggressiveness. 
Mercury accumulation in the Brain is one of the primary causes of Alzheimer’s Disease. 
Exposure to Mercury can cause Anxiety. 
Exposure to Mercury can cause Apathy. 
Exposure to Mercury can cause decreased Attention Span. 
Mercury concentrates in and damages the Brain:
Mercury damages the Blood-Brain Barrier.
Mercury concentrates in and damages the Cerebral Cortex.
Exposure to Mercury can cause impairment of Concentration.
Exposure to Mercury can cause impaired Coordination ability. 
Exposure to Mercury can cause Depression. 
Exposure to Mercury can cause Drowsiness.
Mercury can cause Epilepsy.
Exposure to Mercury can cause Hallucinations. 
Exposure to Mercury (or its vapors) can cause Headache.
Exposure to Mercury (or its vapors) can cause Insomnia. 
Exposure to Mercury can lower Intelligence. 
Exposure to Mercury (or its vapors) can cause Irritability. 
Exposure to Mercury can cause impairment of Learning ability. 
Exposure to Mercury (or its vapors) can cause Memory impairment. 
Mental Retardation can occur as a result of excessive exposure to Mercury. 
Exposure to Mercury is associated with poor (“bad”) Mood. 
Mercury can cause Multiple Sclerosis (MS). 
Exposure to Mercury can cause Nervousness. 
Mercury can cause Numbness.
Exposure to Mercury can cause Paralysis. 
Exposure to Mercury can cause Speech Impairment (in the form of slurred Speech). 
Exposure to Mercury can cause Tremor. 

Respiratory System
Mercury concentrates in and damages the Lungs.

Sexual System
Exposure to Mercury during Pregnancy increases the risk of Birth Defects.
Long-term exposure to Mercury can cause Female Infertility. 
Long-term exposure to Mercury can cause Male Infertility. 

Skin
Mercury toxicity can be an underlying cause of Dermatitis.

Sincerely,

Dr. Michael