Burzynski’s Antineoplastons Hold the First Cures in History of Childhood Brain Cancer 
from burzynskimovie.com

Linda’s comments:  We need to all step to the plate on the pesticide issue in our country.  Other countries have stepped to the plant and banned products that the US continues to carry, thanks to Monsanto…UGHhh

How do we stop this problem…simple, we refuse to purchase these products and when it hits the bottom line we will start winning.

 Link: http://www.e360.yale.edu/content/feature.msp?id=2228

Excerpt:

In the past dozen years, three new diseases have decimated populations of amphibians, honeybees, and — most recently — bats. Increasingly, scientists suspect that low-level exposure to pesticides could be contributing to this rash of epidemics.

by sonia shah

Ever since Olga Owen Huckins shared the spectacle of a yard full of dead, DDT-poisoned birds with her friend Rachel Carson in 1958, scientists have been tracking the dramatic toll on wildlife of a planet awash in pesticides. Today, drips and puffs of pesticides surround us everywhere, contaminating 90 percent of the nation’s major rivers and streams, more than 80 percent of sampled fish, and one-third of the nation’s aquifers. According to the U.S. Fish and Wildlife Service, fish and birds that unsuspectingly expose themselves to this chemical soup die by the millions every year.

This is getting worse folks….did you know that talking on your cell phone while in a car makes it even worse?  This is also another source of radiation.  This study is very enlightening….

Link: http://newsvote.bbc.co.uk/mpapps/pagetools/print/news.bbc.co.uk/2/hi/health/8443541.stm?ad=1

Excerpt:

After all the concern over possible damage to health from using mobile phones, scientists have found a potential benefit from radiation.

Their work has been carried out on mice, but it suggests mobiles might protect against Alzheimer’s.

Linda’s comment:  FINALLY a doctor that gets it…..

Excerpt:

Today, clinicians have major concerns with Toxic Body Burden, consisting of toxic heavy metals, pesticides, volatile organic compounds (VOC’s) and pathogen load, and how these poisons adversely affect the nutritional status of their patients. A particularly critical example of the interference of toxins with nutritional status and biochemical function is the competitive aspect of Mercury and Magnesium.     

Mercury, Magnesium and Adenosine triphosphate (ATP)

Mercury specifically competes with Magnesium and interferes with all Magnesium-dependent metabolic pathways, such as production of energy from ATP and GTP, which directly leads to lack of chemical energy. Every cell in the body requires chemical energy derived from ATP or GTP to function, heal and regenerate. Adenosine-5′-triphosphate (ATP) is a multifunctional nucleotide that is critical as the “molecular currency” of intracellular energy transfer. In this role, ATP transports chemical energy within cells for metabolism. It is produced as an energy source during the processes of photosynthesis and cellular respiration and consumed by many enzymes and a multitude of cellular processes, including biosynthetic reactions, motility and cell division. In signal transduction pathways, ATP is used as a substrate by kinases that phosphorylate proteins and lipids, as well as by adenylate cyclase, which uses ATP to produce the second messenger molecule cyclic AMP. If Mercury is present, this cannot occur.

Genetics also influences our ability to withstand the chemicals we are exposed to, as in this research on Parkinson’s disease. Remember some can eat fish and the mercury they consume clears in as little as 40 days whereas others take nearly 6 months to clear the same dose of mercury.

We have proof from Harvard that the lower level of lead in bones reduces dramatically the incidence of death from heart disease (i.e. high levels have 6 times the death rate) so this is a life time challenge we all face now that we have polluted our earth.

From the aDepartment of Epidemiology, UCLA School of Public Health, Los Angeles, CA; bDepartment of Environmental Health Sciences, School of Public Health, UC Berkeley, CA; cDepartment of Preventive Medicine, Keck School of Medicine, University of Southern California, Los Angeles, CA; dDepartment of Neurology, UCLA School of Medicine, Los Angeles, CA; and eDepartment of Environmental Health Sciences, Center for Occupational and Environmental Health (COEH), UCLA School of Public Health, Los Angeles, CA.

BACKGROUND:: Human, animal and cell models support a role for pesticides in the etiology of Parkinson disease. Susceptibility to pesticides may be modified by genetic variants of xenobiotic enzymes, such as paraoxonase, that play a role in metabolizing some organophosphates. 

METHODS:: We examined associations between Parkinson disease and the organophosphates diazinon, chlorpyrifos, and parathion, and the influence of a functional polymorphism at position 55 in the coding region of the PON1 gene (PON1-55). From 1 January 2001 through 1 January 2008, we recruited 351 incident cases and 363 controls from 3 rural California counties in a population-based case-control study. Participants provided a DNA sample, and residential exposure to organophosphates was determined from pesticide usage reports and a geographic information system (GIS) approach. We assessed the main effects of both genes and pesticides in unconditional logistic regression analyses, and evaluated the effect of carrying a PON1-55 MM variant on estimates of effects for diazinon, chlorpyrifos, and parathion exposures. 

RESULTS:: Carriers of the variant MM PON1-55 genotype exposed to organophosphates exhibited a greater than 2-fold increase in Parkinson disease risk compared with persons who had the wildtype or heterozygous genotype and no exposure (for diazinon, odds ratio = 2.2 [95% confidence interval = 1.1-4.5]; for chlorpyrifos, 2.6 [1.3-5.4]). The effect estimate for chlorpyrifos, was more pronounced in younger-onset cases and controls (</=60 years) (5.3 [1.7-16]). No increase in risk was noted for parathion. 

CONCLUSION:: The increase in risk we observed among PON1-55 variant carriers for specific organophosphates metabolized by PON1 underscores the importance of considering susceptibility factors when studying environmental exposures in Parkinson disease.

Full article: a new report 

Excerpt:

Some carcinogens you already know and fear: cigarettes, asbestos, smoked meat.

But what about the ones you’ve never even heard of? That’s the crux of a new report from theAmerican Cancer Society (ACS), which rounds up 20 “suspected carcinogens” the organization would like to see studied more extensively.

Of course, that research, if it happens, will come after the chemicals, ingredients — and even lifestyle choices — are already embedded into the bedrock of our 24/7 economy.

“The objectives of this report are to identify research gaps and needs for 20 agents prioritized for review based on evidence of widespread human exposures and potential carcinogenicity in animals or humans,” Elizabeth Ward, the co-author of the report, said. 

So just what are these potential cancer causers lurking in our everyday environs? Surge Desk runs down five (not so awesome) favorites.

1. Styrene

Styrene, and its chemical compound colleague styrene-7,-8-oxide, threaten to be a stoner’s worst nightmare. Although only weakly linked to cancer in humans, there’s enough evidence in animal studies to earn the compounds a spot in the top 20.

Where will you find it? Cigarettes, marijuana and leeching into your midnight munchies via foam food packaging. 

How can you avoid it? If you eat, this is one urine test you’re doomed to fail. Styrene was detected in the urine of 87 percent of study participants during a Centers for Disease Control and Prevention trial last year.  

Dr. Arpad Pusztai was an enthusiastic supporter of genetic engineering working on cutting edge safety research, until he discovered the dangerous truth exactly twelve years ago. In Part 1, Jeffrey Smith recounts his dramatic story and why we all owe Dr. Pusztai a debt of gratitude.

Exposing Biotech Propaganda

And don’t miss Part 2, where Jeffrey responds point-by-point to the biotech industry’s spin of the Pusztai affair, which is still being hyped in their new attack website. This is the first in a series of rebuttals to the website’s attack of Genetic Roulette (as promised).

Read the Blog

Objective: This study aimed to investigate antimicrobial treatment of an infected cochlear implant, undertaken in an attempt to salvage the infected device.

Methods: We used the broth microdilution method to assess the susceptibility of meticillin-sensitive Staphylococcus aureus isolate, cultured from an infected cochlear implant, to common antimicrobial agents as well as to novel agents such as tea tree oil. To better simulate in vivo conditions, where bacteria grow as microcolonies encased in glycocalyx, the bactericidal activity of selected antimicrobial agents against the isolate growing in biofilm were also compared.

Results: When grown planktonically, the S aureus isolate was susceptible to 17 of the 18 antimicrobials tested. However, when grown in biofilm, it was resistant to all conventional antimicrobials. In contrast, 5 per cent tea tree oil completely eradicated the biofilm following exposure for 1 hour.

In its never-ending attempt to fabricate “mental disorders” out of every human activity, the psychiatric industry is now pushing the most ridiculous disease they’ve invented yet: Healthy eating disorder.

This is no joke: If you focus on eating healthy foods, you’re “mentally diseased” and probably need some sort of chemical treatment involving powerful psychotropic drugs. The Guardian newspaper reports, “Fixation with healthy eating can be sign of serious psychological disorder” and goes on to claim this “disease” is called orthorexia nervosa — which is basically just Latin for “nervous about correct eating.”

But they can’t just called it “nervous healthy eating disorder” because that doesn’t sound like they know what they’re talking about. So they translate it into Latin where it sounds smart (even though it isn’t). That’s where most disease names come from: Doctors just describe the symptoms they see with a name like osteoporosis (which means “bones with holes in them”).

Getting back to this fabricated “orthorexia” disease, the Guardian goes on to report, “Orthorexics commonly have rigid rules around eating. Refusing to touch sugar, salt, caffeine, alcohol, wheat, gluten, yeast, soya, corn and dairy foods is just the start of their diet restrictions. Any foods that have come into contact with pesticides, herbicides or contain artificial additives are also out.”

Wait a second. So attempting to avoid chemicals, dairy, soy and sugar now makes you a mental health patient? Yep. According to these experts. If you actually take special care to avoid pesticides, herbicides and genetically modified ingredients like soy and sugar, there’s something wrong with you.

But did you notice that eating junk food is assumed to be “normal?” If you eat processed junk foods laced with synthetic chemicals, that’s okay with them. The mental patients are the ones who choose organic, natural foods, apparently.

What is “normal” when it comes to foods?
I told you this was coming. Years ago, I warned NaturalNews readers that an attempt might soon be under way to outlaw broccoli because of its anti-cancer phytonutrients. This mental health assault on health-conscious consumers is part of that agenda. It’s an effort to marginalize healthy eaters by declaring them to be mentally unstable and therefore justify carting them off to mental institutions where they will be injected with psychiatric drugs and fed institutional food that’s all processed, dead and full of toxic chemicals.

The Guardian even goes to the ridiculous extreme of saying, “The obsession about which foods are “good” and which are “bad” means orthorexics can end up malnourished.”