By Linda on Feb 10, 2011 in Infections | comments(0)
Excerpt:
BACKGROUND AND PURPOSE
Neuroborreliosis is a rare cause of stroke in children. We aim here to demonstrate the diagnostic value of gadolinium-enhanced magnetic resonance imaging (MRI) for demonstrating vessel wall abnormality in a child with brainstem stroke.
RESULTS
We report here the case of an 8-year-old boy with cerebral vasculitis and stroke due to Lyme neuroborreliosis. Imaging studies revealed the presence of ischemic lesions in the pons and cerebellum, with focal stenosis of the basilar artery on magnetic resonance angiography and focal gadolinium enhancement of the basilar artery wall. Nine months after treatment, clinical outcome was favorable, with no enhancement of the basilar artery.
CONCLUSIONS
Gadolinium-enhanced MRI provided additional information facilitating the diagnosis of vasculitis in a child with Lyme neuroborreliosis and stroke. The location of vessel wall enhancement was correlated with the topography of the acute infarct, and the lack of vessel lumen obstruction supported the diagnosis of vasculitis rather than any other cause.
By Linda on Sep 2, 2010 in F.I.G.H.T. | comments(0)
Please stop patients from using massive doses of calcium now. The patients all have too much calcium in vascular tissue and now we have data showing that giving calcium is increasing heart disease. And, if possible, try to always administer calcium supplementation with EQUAL AMOUNTS of MAGNESIUM, as I have been teaching for over 20 years now.
Also please realize I use calcium in well over 90 % of my patients. It is in BAM, my multiple called Beyond Any Multiple. I usually never use more than 500 mg of calcium a day for my long-term patients, as that is all most will need since that is the amount we need to offset the excess of phosphorus over calcium in the American diet. This amount will help lower the tendency for the body to develop secondary hyperparathyroidism, which is how the body responds to a chronic dietary intake of more phosphorus than calcium.
Excerpt:
Results 15 trials were eligible for inclusion, five with patient level data (8151 participants, median follow-up 3.6 years, interquartile range 2.7-4.3 years) and 11 with trial level data (11 921 participants, mean duration 4.0 years). In the five studies contributing patient level data, 143 people allocated to calcium had a myocardial infarction compared with 111 allocated to placebo (hazard ratio 1.31, 95% confidence interval 1.02 to 1.67, P=0.035). Non-significant increases occurred in the incidence of stroke (1.20, 0.96 to 1.50, P=0.11), the composite end point of myocardial infarction, stroke, or sudden death (1.18, 1.00 to 1.39, P=0.057), and death (1.09, 0.96 to 1.23, P=0.18). The meta-analysis of trial level data showed similar results: 296 people had a myocardial infarction (166 allocated to calcium, 130 to placebo), with an increased incidence of myocardial infarction in those allocated to calcium (pooled relative risk 1.27, 95% confidence interval 1.01 to 1.59, P=0.038).
Conclusions Calcium supplements (without coadministered vitamin D) are associated with an increased risk of myocardial infarction. As calcium supplements are widely used these modest increases in risk of cardiovascular disease might translate into a large burden of disease in the population. A reassessment of the role of calcium supplements in the management of osteoporosis is warranted.
By Linda on Jul 11, 2010 in Infections | comments(0)
Excerpt:
The significant association of plaque Lp-PLA2 with plaque macrophages and C. pneumoniae suggests an interactive role in accelerating inflammation in atherosclerosis. A possible mechanism for C. pneumoniaein the atherogenic process may involve infection of macrophages that induce Lp-PLA2 production leading to upregulation of inflammatory mediators in plaque tissue. Additional in vitro and in vivo research will be needed to advance our understanding of specific C. pneumoniae and Lp-PLA2 interactions in atherosclerosis.
By Linda on May 17, 2010 in F.I.G.H.T., Infections | comments(0)
Excerpt:
If you have multiple sclerosis (MS)––or you know someone who does––you probably remember how long it took to make the diagnosis. You also may remember a lot of blood tests, a lumbar puncture, at least one magnetic resonance imaging (MRI) scan, as well as many visits and examinations by various doctors. You may wonder why it still takes so long to make the diagnosis in this modern age of MRIs and other sophisticated tests. We are going to try to explain why it can be so difficult for even the most expert MS neurologist to determine that someone has MS. You have to live with the diagnosis and face the disease and the treatments. You should understand and have confidence in the diagnosis. Also, if your case of MS does not fit the typical pattern, you need to be aware of the other disorders that can mimic MS. This is important because the treatments may be very different and, just as in most cases of MS, treatment begun early in the course of the disease is the best way to prevent or slow further neurologic damage.
MRI and new laboratory tests have definitely helped speed the diagnosis, but it still takes longer than anyone would wish, even in easy cases. This is partly because of the variable nature of the disease in its many signs and symptoms. But it is also because a rather long list of other medical disorders can cause neurologic symptoms and signs that resemble MS. Furthermore, the “white spots” on brain MRI can be caused by a number of other conditions that also need to be ruled out.
The diagnosis of clinically definite MS requires that a person experience at least two neurologic symptoms of the type seen in MS, in two different areas of the central nervous system (CNS), at two different times (‘disseminated in space and time’). Most typically, the symptoms are optic neuritis plus either an abnormal sensation or a problem with movement. It can also be numbness in one part of the body and weakness or lack of coordination in another. But in every case, there can be no other explanation for the symptoms, the changes seen on the MRI, and the abnormalities in the spinal fluid. Many “mimics” need to be ruled out in order to make the diagnosis of MS.
By Linda on Mar 15, 2010 in General News | comments(0)
Linda’s comments: Finally a university does a study and admits they find that Fiber boosts our immune systems. AND yes, a good Fiber can reduce inflammation associated with obesity related diseases. The BEYOND FIBER I take has more than just fiber….please go here and read about the fiber I take twice daily… www.longevityplus.com <BEYOND FIBER> This Beyond Fiber is part of the FIGHT protocol that I have been on for over 1 1/2 years. I had tried over 15 different fibers before Beyond FIber. I will never go back. Healing our cells helps us to recover faster from different infections.
Join the FIGHT team and get healthy!! Go to www.gordonresearch.com and find the Webinar’s on the F.I.G.H.T. protocol. I do believe you will be pleasantly surprised what you learn there. By reducing our total body burden of pathogens and toxins leads us to a healthier life and a happier YOU.
Excerpt:
A new University of Illinois study touts the benefits of soluble fiber – found in oats, apples, and nuts, for starters – saying that it reduces the inflammation associated with obesity-related diseases and strengthens the immune system.
Continued
By Linda on Jan 26, 2010 in Food | comments(0)
Full article: http://www.orthomolecular.org/resources/omns/v06n02.shtml
Excerpt:
How To Destroy Confidence In Vitamins When You Do Not Have The Facts
(OMNS, January 11, 2010) “Ladies and Gentlemen, welcome to this year’s annual meeting of the World Headquarters Of Pharmaceutical Politicians, Educators, and Reporters (WHOPPER).
“Let us get right to the point. Many of our members and affiliates have complained about what is, for us, an alarming and dangerous segment of health care: so-called ‘orthomolecular medicine.’ We wish to assure you, although this therapeutic approach is, unfortunately, very effective in preventing and treating disease, that we will make sure the public will never learn of it. We can say this with considerable confidence, since for over 50 years we have managed to keep virtually all psychiatrists from using niacin to treat schizophrenia; we have kept cardiologists from prescribing vitamin E and co enzyme Q10 for heart disease; and we have kept general practitioners from prescribing vitamin C for viral illnesses.
“Yes, it has really been a triumphant half-century. How did we do it? It is really quite easy. Here is a summary for those of you that may have missed the last WHOPPER meeting.
By Linda on Jan 22, 2010 in Food | comments(0)
Linda’s comments: Poor nutrition will and does cause many illness in adults and children. Eating GMO foods, fast foods, junk foods, drinking any kind of soda regular and/or diet you are asking for troble. For those young women out there who are thinking about having children, you need to think first about cleaning up your body before getting pregnant. An unborn baby is a toxic dump for the mother. All the toxins and bad things she eats go right to the baby. If the mother has amalgam fillings all that mercury goes straight to the unborn child.
While diet/nutrition is one of the hardest things in the world it is one of the most important things for good health. Today, we have to worry about all the GMO foods. We have to be concerned about pesticides, herbicides, how it is prepared, packaged and stored before it gets to your mouth.
Using drugs to fix the problems that a poor diet has caused is only adding more problems to the mix. Why add more toxins to our bodies. Try cleaning up your lifestyle, and adding vitamins, herbals and supplements to your everyday living.
Focus on cleaning out the toxins you have packed in and stored by getting on a lifelong daily detox, them supplementing the nutrients using a good quality supplement product. Using drugs will only compound your problems.
Regards,
Linda or Angel
Excerpt:
(OMNS, November 13, 2008) A recent study suggested that statins might be used to avoid the effects of nutritional deficiency. Writing in the New England Journal of Medicine, the Jupiter group described a study of statin drugs in people with high C-reactive protein and low cholesterol. (1) High C-reactive protein levels are associated with inflammation and heart disease/stroke. The authors concluded that, in apparently healthy persons with elevated C-reactive protein levels, rosuvastatin (Crestor) significantly reduced the incidence of major cardiovascular events. Continued
By Linda on Nov 29, 2009 in Infections | comments(0)
Encephalopathy is like fine art: Most people know it when they see it, but there is very little agreement on how to define it. At the 14th International Lyme Disease Conference, Brian A. Fallon, MD,[1] of Columbia University and the New York State Psychiatric Institute, New York, NY, tried to do just that. More importantly, he described the different ways one can define encephalopathy, the strengths and limitations of each approach, and significantly, what other aspects of life can give the impression of encephalopathy where none exists. First, one must evaluate patients with persistent Lyme encephalopathy by asking the following questions:
Is the diagnosis correct?
Are there comorbid psychiatric disorders that could be treated better? Does the patient have a psychogenic medical illness? What was the patient’s response to prior antibiotics?
Was previous treatment adequate? How long was the course, and what was the route of administration? Was there a subsequent relapse Continued
By Linda on Nov 29, 2009 in Infections | comments(0)
Encephalopathy is like fine art: Most people know it when they see it, but there is very little agreement on how to define it. At the 14th International Lyme Disease Conference, Brian A. Fallon, MD,[1] of Columbia University and the New York State Psychiatric Institute, New York, NY, tried to do just that. More importantly, he described the different ways one can define encephalopathy, the strengths and limitations of each approach, and significantly, what other aspects of life can give the impression of encephalopathy where none exists.
First, one must evaluate patients with persistent Lyme encephalopathy by asking the following questions:
Is the diagnosis correct?
Are there comorbid psychiatric disorders that could be treated better? Does the patient have a psychogenic medical illness? What was the patient’s response to prior antibiotics?
Was previous treatment adequate? How long was the course, and what was the route of administration? Was there a subsequent relapse Continued
