People face prison over shaken baby syndrome yet it now appears that many of the cases are Vitamin D deficiency during pregnancy. This is very sad to read and I doubt if many of you are knowledgeable about these facts:
broken bones and bleeding eyes could happen during a vaginal delivery, but what about brain hemorrhage, certainly that is proof of abuse? Well, it has been proof – proof positive of parental child abuse – for decades, sending thousands of parents to prison for “Shaken Baby Syndrome.” Guess what, it turns out that 26% of newborn infants have brain hemorrhages after a normal, natural, vaginal delivery.
This is information that you need to know, as perfectly innocent parents are being punished because no one is measuring Vitamin D levels during pregnancy or putting this together with “temporary brittle bone disease,” not “shaken baby syndrome and no one is diagnosing rickets in newborn children – yet here it is.”
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Dear Dr. Cannell:
I am writing to you today to see if there is any information or help you can give to me. On October 25th of 2009, I gave birth to our wonderful son & we were overjoyed. Four days later we noticed that our son had a swollen right thigh so we took him back to the hospital & there an x-ray was done & he had a fractured femur!
Then on the 2nd of Nov. the hospital carried out further scans & we were told that he had fractures to the fibula, tibia & 3 fractures of the ribs. You can now only imagine the situation we are now in but we remain strong & are fighting this all the way knowing that we have not shaken our baby or abused our son or caused these fractures. It was only after our repeated requests that the hospital carried out blood tests on our son for calcium, Vitamin D etc. this was after him been in the hospital & after we had been accused of Shaken Baby Syndrome.” These tests came back as normal.
The impact of intensive lipid management, omega-3 fatty acid, and vitamin D3 supplementation on atherosclerotic plaque was assessed through serial computed tomography coronary calcium scoring (CCS). Low-density lipoprotein cholesterol reduction with statin therapy has not been shown to reduce or slow progression of serial CCS in several recent studies, casting doubt on the usefulness of this approach for tracking atherosclerotic progression. In an open-label study, 45 male and female subjects with CCS of > or = 50 without symptoms of heart disease were treated with statin therapy, niacin, and omega-3 fatty acid supplementation to achieve low-density lipoprotein cholesterol and triglycerides < or = 60 mg/dL; high-density lipoprotein > or = 60 mg/dL; and vitamin D3 supplementation to achieve serum levels of > or = 50 ng/mL 25(OH) vitamin D, in addition to diet advice. Lipid profiles of subjects were significantly changed as follows: total cholesterol -24%, low-density lipoprotein -41%; triglycerides -42%, high-density lipoprotein +19%, and mean serum 25(OH) vitamin D levels +83%. After a mean of 18 months, 20 subjects experienced decrease in CCS with mean change of -14.5% (range 0% to -64%); 22 subjects experienced no change or slow annual rate of CCS increase of +12% (range 1%-29%). Only 3 subjects experienced annual CCS progression exceeding 29% (44%-71%). Despite wide variation in response, substantial reduction of CCS was achieved in 44% of subjects and slowed plaque growth in 49% of the subjects applying a broad treatment program.
Join Dr. Garry F. Gordon as he explains the incredible burden that environmental toxins place on our body.
He will also explain proven protocols for detoxification.
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F.I.G.H.T. for Your Health T is for Toxins
Date:
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Clinical treatment failures have been reported to occur in early Lyme borreliosis (LB) for many suitable antimicrobial agents. Investigations of possible resistance mechanisms of the Borrelia burgdorferi complex must analyze clinical isolates obtained from LB patients, despite their receiving antibiotic treatment. Here, borrelial isolates obtained from five patients with erythema migrans (EM) before the start of antibiotic therapy and again after the conclusion of treatment were investigated. The 10 isolates were characterized by restriction fragment length polymorphism analysis and plasmid profile analysis and subjected to susceptibility testing against a variety of antimicrobial agents including those used for initial chemotherapy. Four out of five patients were infected by the same genospecies (Borrelia afzelii, n = 3; Borrelia garinii, n = 1) at the site of the EM lesion before and after antimicrobial therapy. In one patient the genospecies of the initial isolate (B. afzelii) differed from that of the follow-up isolate (B. garinii). No significant changes in the in vitro susceptibilities became obvious for corresponding clinical isolates before the start and after the conclusion of antimicrobial therapy. This holds true for the antimicrobial agents used for specific chemotherapy of the patients, as well as for any of the additional agents tested in vitro.
A 16-year-old male with headache, vomiting, fever, neck stiffness, and a positive Kerning’s sign was referred to Boali Medical Center. The cerebrospinal fluid examination revealed a lymphocytic meningitis, and the blood smears was positive for Borrelia. He was successfully treated with doxycycline
Full article: http://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pu
bmed&id=20350056&retmode=ref&cmd=prlinks
Excerpt:
Dr. Robert Lane received a B.A. degree in psychology from the
University of California at Berkeley (UCB), an M.A. degree in
biology at San Francisco State College, and a Ph.D. in entomology
at UCB. While employed as a California State public health
biologist he began his long-standing studies of the biology of
ticks and the ecology and epidemiology of tick-borne disease
agents. In 1984, Dr. Lane joined the faculty of UCB as a medical
entomologist, a position he has held until the present. The
diseases he and his many co-workers have investigated include
Colorado tick fever, human granulocytic anaplasmosis, relapsing
fever, Rocky Mountain spotted fever, tularemia, and particularly
Lyme disease. Findings from these studies have elucidated the
basic transmission cycles of and risk factors for spotted
fever-group rickettsiae and Lyme disease spirochetes in the far
western United States. Bob is a Fellow of both the California
Academy of Sciences and the American Association for the
Advancement of Science, a recipient of a UCB Biology Faculty
Research Award and the C.W.
Linda’s comments: What we use on our bodies is extremely important. As we all know it is very important to reducing our exposure to pathogens, toxic toxins and other chemicals used in our bathrooms. We are being slammed with way to many chemical today, which doesn’t help those of us with Chronic illness. Plus, Lyme patients don’t need to be feeding the critters, which LOVE these toxic chemicals.
Is it time to put your skin on an organic diet? Soap, shampoo, lotions, toothpaste, cosmetics and other personal care items are full of chemicals, some of which are toxic. Absorbed through the pores, these chemicals enter the bloodstream.
Genuinely organic products, though, are hard to find. While the federal government enforces strict standards for organic food, it has no guidelines for “organic” personal care items.
That means the word “organic” on a label is often “meaningless marketing shtick,” said David Bronner, president of Escondido-based Dr. Bronner’s Magic Soaps.
The authors present the case of a young man with aplastic anemia who went into shock and died after several red blood cell unit transfusions. Immunohematological studies did not show any abnormality and blood cultures from patients and blood bags were negative. The ultrastructural findings, allied with current scientific knowledge, permitted the diagnosis of Bartonella sp. infection. In face of this diagnosis, two possibilities should be considered: the first one is that the patient was already infected by the bacteria before the last RBC unit transfusion. The pathogen could be involved in aplastic anemia etiology and in the failure to recover hemoglobin levels, in spite of the transfusions. The second possibility is that the RBC unit was contaminated with a Bartonella sp., which would have led to a state of shock, causing the death of the patient.
A review of studies of maternal exposure to infectious agents and schizophrenia in their offspring suggests that eliminating certain infections could prevent as many as 30 percent of schizophrenia cases.
Call it an instance of science being stranger than science fiction, and of nature’s unintended consequences.
In 2000, a team of British researchers published a remarkable paper in the journal of the Royal Society titled “Fatal Attraction in Rats Affected With Toxoplasma Gondii.” It seems that rodents infected with Toxoplasmosis gondi (T. gondii), a parasite that normally thrives in cats, become fatally attracted to cat urine, causing them to shed their normal avoidant behavior in the presence of a cat.
Naturally, cat catches rodent and devours it, with the result that the parasite T. gondii is again where it belongs: in a feline host.
Observing this case of attraction gone fatally wrong, the researchers, from the University of Oxford, postulated that T. gondii, one of nature’s most successful organisms, had developed an ingenious evolutionary mechanism for manipulating the behavior of the rodent—in whom the parasite would have reached a dead end—so that the rodent seeks out, suicidally, the feline host in which the parasite can thrive and complete its life cycle.
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