Oral Vitamin K-3, with comments by Dr. Gordon
By Linda on Feb 22, 2011 in Food | comments(0)
Could oral Vitamin K-3 activate oral Vitamin C so efficiently that it could be a viable alternative to IV Vitamin C for seriously ill patients needing oxidative therapies? In Vitro research is very promising and I will present more on this topic for the ACAM oxidative workshop mid-April in Minneapolis that I am doing with Robert Rowen and others.
Now there are IN VIVO human trials so read the entire report attached and learn much more! This is vital to saving lives. Many patients have been led to believe that nothing but IV Vitamin C can help extend their lives. Whereas we know that those that keep their Vitamin C urine test strips in the bright yellow range prove that they have high levels of vitamin C in their body at all times 24/7. By using the special vitamin C delivery system called BIOEN’R-G’Y C they seem to always live much longer than anyone expected. It now seems that they might add oral Vitamin K-3 and do even better!
This is a key document like the new Johnson And Johnson blood test for cancer followed always with a reference to Kobayashi and the tamoxifen/radiation article on breast cancer and the Harvard Study on telomeres and age reversal in old mice with tamoxifen, which have all been shared with you on FACT.
Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com
Excerpt:
The Vitamin C:Vitamin K3 System – Enhancers and Inhibitors of the Anticancer Effect
Davis W. Lamson, MS, ND; Yu-Huan Gu, PhD; Steven M. Plaza, ND, LAc; Matthew S. Brignall, ND; Cathy A. Brinton, ND; Angela E. Sadlon, ND
Abstract
The oxidizing anticancer system of vitamin C and vitamin K3 (VC:VK3, producing hydrogen peroxide via superoxide) was combined individually with melatonin, curcumin, quercetin, or cholecalciferol (VD3) to determine interactions. Substrates were LNCaP and PC-3 prostate cancer cell lines. Three of the tested antioxidants displayed differences in cell line cytotoxicity.Melatonin combined with VC:VK3 quenched the oxidizing effect, while VC:VK3 applied 24 hours after melatonin showed no quenching. With increasing curcumin concentrations, an apparent combined effect of VC:VK3 and curcumin occurred in LNCaP cells, but not PC-3 cells. Quercetin alone was cytotoxic on both cell lines, but demonstrated an additional 50-percent cytotoxicity on PC-3 cells when combined with VC:VK3. VD3 was effective against both cell lines, with more effect on PC-3.
This effect was negated on LNCaP cells with the addition of VC:VK3. In conclusion, a natural antioxidant can enhance or decrease the cytotoxicity of an oxidizing anticancer system invitro, but generalizations about antioxidants cannot be made.
