(2004) I am a member of the Chelation Discussion Group, headed by the guru of Chelation therapy, Garry Gordon, M.D., D.O. It may have well over 2000 health professional members now on a worldwide basis. The theme seems to be primarily questions answered by Dr. Gordon as well as astute contributions from the members. Well, there is a tremendous number of communications per week. I used to print them all out and save them, but now I have decided to record the essence of many of the teachings in my brain.
Parapoxviruses are a genus of the double-stranded DNA family of poxviruses that infect ruminants, and zoonotic transmission to humans often results from occupational exposures. Parapoxvirus infection in humans begins with an incubation period of 3 to 7 days, followed by the development of one or more erythematous maculopapular lesions that evolve over the course of several weeks into nodules. In 2009, parapoxvirus infection was diagnosed in two deer hunters in the eastern United States after the hunters had field-dressed white-tailed deer. We describe the clinical and pathological features of these infections and the phylogenetic relationship of a unique strain of parapoxvirus to other parapoxviruses. Deer populations continue to increase, leading to the possibility that there will be more deer-associated parapoxvirus infections.
To determine the cause of spotted fever cases in the southern United States, we screened Gulf Coast ticks (Amblyomma maculatum) collected in Arkansas for rickettsiae. Of the screened ticks, 30% had PCR amplicons consistent with Rickettsia parkeri or Candidatus Rickettsia amblyommii.
The Centers for Disease Control and Prevention identified Arkansas as a leading state for the incidence of Rocky Mountain spotted fever (causative agent Rickettsia rickettsii) and reported >15 cases per 1,000,000 persons in 2002 (1). Given the known cross-reactivity of serologic testing results for spotted fever group (SFG) rickettsia, it is unclear if cases outside the natural range of the vectors for R. rickettsii are misdiagnosed, if the pathogen is less virulent than previously suggested, or if additional rickettsiae are responsible for pathogenesis (2).
*********They had to spend thousands perhaps, even more to find that Birds are playing an important role in the spread of Lyme Disease?? What??? Are you telling me that birds stop at each border and request the right to fly over your state?? What a big waste of money….why doesn’t Yale and the Infectious Disease docs and other specialists at Yale, who say there is NO chronic Lyme, spend money finding a cure for Lyme disease instead of finding out what we patient’s already know. Give me a break!!
*********It is time that patient’s start standing up and pushing back….sitting back and doing nothing is getting us no where.
*********We need to stop “fearing” city hall, when we can be city hall in these Lyme wars.
*********Just remember folks, Lyme isn’t just carried by ticks….look to birds, rodents, mice, we need to understand that it is up to us Lymie’s to take a stand.
Regards,
Linda
Article Excerpt:
New Haven, Conn. – The range of Lyme disease is spreading in North America and it appears that birds play a significant role by transporting the Lyme disease bacterium over long distances, a new study by the Yale School of Public Health has found. The study appears online in the journal Frontiers in Ecology and the Environment.
Researchers analyzed published records and concluded that at least 70 species of North American birds are susceptible to infection by black-legged ticks (Ixodes scapularis), the principal vector of the Lyme disease bacterium (Borrelia burgdorferi). The evidence also suggests that these bird species are dispersing infected ticks into areas that had previously been free of the disease, such as Canada.
Lyme disease bacterium is usually associated with small mammals such as mice and squirrels. Immature ticks (in the larval and nymphal stages) become infected with the bacterium when they feed on these mammals. During subsequent blood meals, an infected tick transmits the infection to other hosts, including humans. White-tailed deer-while playing an important role in maintaining and spreading tick populations-are a biological dead end for the bacterium because its blood is immune to infection.
Birds, however, are not immune and numerous species get infected and are capable of transmitting the pathogen onto ticks, the researchers found. What remains to be seen is whether the B. burgdorferi strains that can infect birds can also cause disease in humans. If so, the role of birds in the epidemiology of Lyme disease could be profound.
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