By Linda on Sep 29, 2010 in Toxins | comments(0)
Dear Sherrel,
Thank you for your note. As I told you by phone we have been getting
quite a few reactions from Truvia. Pure stevia is fine but Truvia is
made by Coke, and has chemicals. It is sweetened with Erythritol and
has added an ingredient of stevia only. In fact, I did an expose on
it which was shown in France. You can be sure if Coke and Pepsi
have anything to do with sweeteners we have no way of assuring
safety. Here is an email I wrote about
it: http://www.mpwhi.com/health_problems_and_truvia.htm and an
investigation by Arthur Evangelista who use to work for the FDA. Continued
By Linda on Aug 14, 2010 in Toxins | comments(0)
Only for brain tumors in New York and New Jersey. You may wish to
subscribe to the Aspartame Information List on www.mpwhi.com so you
will get copies of my emails and ask questions if you do file suit.
Also, if you need to know anything about the problems let me know and
I’ll answer them for you. For instance in the case of diabetes,
aspartame not only can precipitate diabetes but it simulates and
aggravates diabetic retinopathy and neuropathy, destroys the optic
nerve, causes diabetics to go into convulsions, interacts with
insulin and the free methyl alcohol causes them to lose limbs.
Below my signature is the Aspartame Resource Guide. Aspartame
Awareness Weekend is the first weekend after Labor Day. Continued
By Linda on May 28, 2010 in Infections | comments(0)
Excerpt:
The ability of the thymus to generate a population of T cells that is, for the most part, self-restricted and self-tolerant depends to a great extent on the Ags encountered during differentiation. We recently showed that mycobacteria disseminate to the thymus, which raised the questions of how mycobacteria within the thymus influence T cell differentiation and whether such an effect impacts host-pathogen interactions. Athymic nude mice were reconstituted with thymic grafts from Mycobacterium avium-infected or control noninfected donors. T cells generated from thymi of infected donors seemed generally normal, because they retained the ability to reconstitute the periphery and to respond to unspecific stimuli in vitro as well as to antigenic stimulation with third-party Ags, such as OVA, upon in vivo immunization. However, these cells were unable to mount a protective immune response against a challenge with M. avium. The observation that thymic infection interferes with T cell differentiation, generating T cells that are tolerant to pathogen-specific Ags, is of relevance to understand the immune response during chronic persistent infections. In addition, it has potential implications for the repertoire of T cells generated in patients with a mycobacterial infection recovering from severe lymphopenia, such as patients coinfected with HIV and receiving antiretroviral therapy.
