All Posts Tagged With: "neurology"

Full article: http://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=20561635&retmode=ref&cmd=prlinks

Excerpt:

RESULTS: Eleven patients who presented with acute central nervous
system (CNS) syndrome fulfilled the diagnostic criteria for
neuroborreliosis. In the CSF studies, lymphocytic pleocytosis
with elevation of total protein and normal glucose was noted in 9
patients. The clinical syndromes were confirmed by magnetic
resonance imaging and electrophysiological studies. Ten of 11
patients were found to have brain stem or spinal cord lesions on
neuroimaging studies, and 8 patients had concomitant peripheral
nervous system involvement with electrophysiological evidence of
multiple root lesions with an acute or subacute course.
CONCLUSIONS: Lyme neuroborreliosis presenting as acute CNS
syndrome with peripheral nervous system involvement
simultaneously in the disease course, especially acute
myelopolyradiculitis, in Taiwan is different from Lyme
neuroborreliosis seen in Europe and North America. The treatment
outcome is variable and based on the severity of initial
neurological deficits, early diagnosis, and early management.
Copyright (c) 2010 Elsevier B.V. All rights reserved.

Excerpt:

We report the case of a 30-year-old man who developed severe
dysphagia owing to neuroborreliosis. He showed dysphagia,
diplopia, hiccups, and walking difficulty Neurological
examination revealed mild disturbance of consciousness, diplopia
on left lateral gaze, left-side-dominant blephaloptosis,
gaze-evoked horizontal nystagmus on left lateral gaze, mild
bilateral muscle weakness, palatoplegia, dysphagia, dysarthria,
and truncal ataxia An increased pharyngeal reflex caused
dysphagia in this patient. An EEG revealed intermittent high
amplitude slow wave activity. However, head MRI, blood count,
serum chemistry, and cerebrospinal fluid examination showed no
abnormality. Initially, brainstem encephalitis with unknown
etiology was diagnosed. The hiccups, diplopia, and ptosis were
improved by corticosteroid therapy, but other symptoms were
refractory to corticosteroid therapy and IVIg.

Excerpt:

BACKGROUND: Lyme borreliosis is a multisystemic disease which affects several organs such as skin, nervous system, joints and the heart. The presented study focused on patients with persisting symptoms of the disease, which could be in correlation with Lyme disease but antiborrelial antibodies were not confirmed by screening tests. MATERIAL AND METHODS: 32 patients with anamnestic data and suspected clinical signs of lyme borreliosis were tested for the presence of antiborrelia antibodies by using ELISA and westernblot analysis and the state of cellular and humoral immunity. RESULTS: All patients had specific antiborrelial antibodies confirmed by using the westernblot in spite of negative ELISA. Immunological investigations revealed a deficiency of cellular immunity in all patients and in a part of them (15.6%) a deficiency of humoral immunity was also found. The presence of different types of autoantibodies was detected in 17 (53.1%) patients. CONCLUSION: In patients with persisting difficulties that could be associated with Lyme disease, it is necessary to use the westernblot test which could prove the presence of specific antibodies. It is probably due to the very low production of specific antibodies caused also by the status of immune deficiency detected in all our patients (Tab. 1, Ref. 11).

Only two spirochetal infections are known to cause nervous system infection and
damage: neurosyphilis and neuroborreliosis (nervous system Lyme disease).
Diagnosis of both generally relies on indirect tools, primarily assessment of
the host immune response to the organism. Reliance on these indirect measures
poses some challenges, particularly as they are imperfect measures of treatment
response. Despite this, both infections are known to be readily curable with
straightforward antimicrobial regimens. The challenge is that, untreated, both
infections can cause progressive nervous system damage. Although this can be
microbiologically cured, the threat of permanent resultant neurologic damage,
often severe in neurosyphilis and usually less so in neuroborreliosis, leads to
considerable concern and emphasizes the need for prevention or early and
accurate diagnosis and treatment.

Linda’s comment:  The FIGHT program is a perfect example of how you can fight Lyme disease.  As Dr Patricia Gerbarg, MD has found out.  She has not experienced the FIGHT program to my knowledge, but the protocols she mentioned are all part of the FIGHT program.  It is a must that we reduce the total body burden of toxins and pathogens to fight the Lyme critters.  Lyme loves heavy metals.  We are slammed every time we walk out our front doors with 500 to 600 environmental toxins.  It is a daily battle, but I found the FIGHT program made this all very easy.  I only wish I had  the knowledge of the FIGHT program with the first Lyme infection.

I have never taken any antibiotics and don’t intend to, but I focused on the lifelong daily detox program FIGHT and cleaned up my lifestyle, home and diet.  I got rid of all the GMO foods, gluten, sugar, caffeine, alcohol and my body shakes if I pass a fast-food restaurant….the smell turns my stomach.  Once you clean  up your diet and clean up your homestead, you begin to feel better….Lifestyle is probably one of the hardest things I have ever done….Yes, I cheat, but at least now I have learned to cheat. and can neutralize a toxic food if I eat it.  Once you begin to feel better it makes the journey of cleaning up much easier. Just give the FIGHT program 90 days and you too will feel the difference.

Regards, Linda Continued