All Posts Tagged With: "infection"

This issue about flame retardant is the tip of the iceberg. Go to Google and search on Ten Americans and find a video to learn what else is in everyone and at a much higher concentration in every newborn. Join me as I try to detox the world using things like Zeolite, high dose oral Vitamin C, Beyond Fiber, Greens, Maca and daily Zeolite starting with ACZ and then move on to ZeoGold, at least one daily for life.

Go to www.gordonresearch.com and watch my webinars on FIGHT and then the hour long specials on FIGHT including Food, Infection, Genetics, Hormones and Heavy Metals and Toxins. I find most health problems are multifactorial and the acronym FIGHT helps remind my friends and patients to leave no stone unturned in their quest for optimal health, as I clearly am enjoying now at last at age 75. Detoxing is a life time program!!

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com

Full article: http://articles.mercola.com/sites/articles/archive/2010/07/06/flame-retardant-causes-altered-thyroid-hormone-levels.aspx

Excerpt:

Pregnant women with higher blood levels of PBDEs, a common class of flame retardants, had altered thyroid hormone levels — a fact that could have implications for fetal health. 
PBDEs, or polybrominated diphenyl ethers, are organobromine compounds which are found in household items such as carpets, electronics and plastics. PBDEs can leach out into the environment and accumulate in human fat cells. 

Eurekalert reports:
“Studies suggest that PBDEs can be found in the blood of up to 97 percent of U.S. residents, and at levels 20 times higher than those of people in Europe. Because of California’s flammability laws, residents in this state have some of the highest exposures to PBDEs in the world.”
Sources:
   Eurekalert June 21, 2010 
   Environmental Health Perspectives June 21, 2010

Full article: http://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=20207197&retmode=ref&cmd=prlinks

Excerpt:

Dogs and people are exposed to and susceptible to infection by
many of the same tick-borne bacterial pathogens in the order
Rickettsiales, including Anaplasma phagocytophilum, Ehrlichia
canis, E. chaffeensis, E. ewingii, Rickettsia rickettsii, R.
conorii, and other spotted fever group rickettsiae. Recent
findings include descriptions of novel Ehrlichia and Rickettsia
species, recognition of the occurrence and clinical significance
of co-infection, and increasing awareness of Rhipicephalus
sanguineus-associated diseases. Newer molecular assays are
available, although renewed efforts to encourage their use are
needed. This review highlights the ecology and epidemiology of
these diseases, and proposes avenues for future investigation.
Published by Elsevier Ltd.

Excerpt:

June 17, 2010 — Concern that a new human gamma-retrovirus may be transmissible through blood has led one infectious diseases specialist to recommend new steps to protect the US blood supply against possible infection with the virus.

The transfusion medicine organization AABB has formed a task force to study the transmission potential of xenotropic murine leukemia virus–related virus (XMRV), which has been linked to familial prostate cancer and, more recently, to chronic fatigue syndrome (CFS).

Full article: http://www.ispub.com/ostia/index.php

Excerpt:

Thrombocytopenia is a common manifestation of all tick-borne diseases. Low platelet numbers contribute significantly towards the morbidity and mortality of infection. However, the pathogenesis of thrombocytopenia in many of the tick-borne diseases is poorly understood. Quantitative changes in platelet counts associated with infection may result from decreased marrow production, hypersplenism, consumption due to widespread endothelial damage or disseminated intravascular coagulation, as well as immune-mediated platelet destruction. Infection-induced thrombocytopenia may infrequently be associated with serious thrombosis. Direct infection of platelets by selected tick-borne pathogens also facilitates their dissemination within the host. This article reviews the mechanisms of thrombocytopenia associated with tick-borne infections, and discusses the therapeutic options available for managing this potentially fatal complication.

Conclusions

Thrombocytopenia due to tick-borne infection is likely to be of multifactorial etiology. In many of these infections, however, the actual mechanism of thrombocytopenia still remains unknown. Iatrogenic causes in some cases, such as drugs, should not be overlooked. Low platelet numbers in infected individuals may not only manifest with increased bleeding, but may herald more widespread life-threatening microthrombus formation. In addition to quantitative platelet disorders, qualitative defects may also be present. For example, inhibition of platelet migration has been induced by serum from E.canis-infected dogs, even before platelet numbers declined and before the appearance of specific humoral
antibodies (Kakoma et al 1978). The release of platelet factor 3, a phospholipid released from activated platelets that is necessary for the intrinsic conversion of prothrombin to thrombin, is also markedly decreased as a result of ehrlichial infection (Pierce et al 1977). Furthermore, platelet aggregation in ehrlichiosis can be prevented by autoantibodies directed against their surface glycoproteins (Lovering et al 1980; Harrus et al 1996).

Therapy for thrombocytopenia requires treatment or removal of the underlying infection, in addition to maintenance of platelet counts and hemostatic function. However, identification and correction of a specific tick-borne infection is only possible if the infection is considered in the differential diagnosis of thrombocytopenia. Rapid treatment of the underlying infection should result in normalization of platelet counts. Ehrlichia, bartonella and RMSF should also be excluded in cases presenting with a clinical picture resembling TTP, particularly in those patients that prove difficult to manage, and in regions where the incidence of tick-borne illness is high. The efficacy of platelet transfusions in many of the tick-borne diseases is unclear and anecdotal (Van Eeden et al 1985).

NOTHING is rare these days!!  If Hepatitis, parasites, Lyme, can be passed on why do they find it so hard to believe that this amoeba can be passed on????  There are liver recipients who get a new liver ONLY to find it is infected with Hepatitis, Lyme and parasites!!  If the health departments will not classify Lyme as an STD, how can we expect them test for parasites.  Yes, I agree there are many parasites that we humans can and DO pass on to each other….Another reason to make sure you are on a life-long detox, so you are aggressively fighting these “rare” issues and disease.  If it is true and you can get the infection by breathing it in, then it is a MUST that we use ACS200ppm. 
 
Regards,
Linda or Angel

JACKSON, Miss. – An extremely rare infection has been passed from an organ donor to at least one recipient in what is thought to be the first human-to-human transfer of the amoeba, medical officials said Friday. Four people in three states received organs from a patient who died at the University of Mississippi Medical Center in November after suffering from neurological problems, said Dave Daigle, a spokesman for the Centers for Disease Controls and Prevention. Continued

Rates of infection of Amblyomma americanum (L.) by Ehrlichia chaffeensis were
compared in 100 ticks collected from sites in each of four states: Indiana,
North Carolina, Kentucky, and Mississippi. The overall infection rates were
similar among sites, ranging from 1 to 4%. Because pathogenic differences may
exist between E. chaffeensis strains, nested polymerase chain reaction (PCR)
amplification of the variable-length PCR target (VLPT), and sequencing of the
amplicons were performed to differentiate between strains. The most common
infecting strains at all sites exhibited a repeat profile of 1,2,3,4
(corresponding to the Arkansas/Jax/Osceola and Liberty strains). To determine
whether the minimum infection rates (MIRs) or the most common infecting strain
were changing over time in southern Indiana sites, 2765 ticks from six counties
in 2000 and 837 ticks from seven counties in 2004 also were examined in pools of
five ticks per pool. The MIRs for 2000 and 2004 were 3.5 and 4.2% respectively,
suggesting that the overall MIRs remained low. At two sites, in Pike and
Harrison counties, however, infection rates more than doubled from 2000 to 2004
(7 to 16% and 0.3 to 2.7% respectively). Across all sites, the most common
infecting strains (Arkansas/Jax/Osceola and Liberty) did not significantly
change (68% in 2000; 79% in 2004). Continued

Human leptospirosis can be a difficult infection to describe, as the
symptoms can vary dramatically between patients. Some symptoms are
extremely common, but only a small number of patients will experience
the severe life-threatening illness known as Weil’s disease. The
severity of the infection depends on the age and general health of the
patient, plus the serovar (strain) of bacteria involved and the number
of bacteria that entered the patient’s body. Continued

MINNEAPOLIS (AP) _ Deer ticks are expanding their range in the Upper Midwest and southern Canada, new ticks are moving into the area and existing ticks are picking up new diseases, increasing the threat of illness to hikers tramping through the region’s woods. Continued

Central Role of the Holliday Junction Helicase RuvAB in vlsE Recombination and Infectivity of Borrelia burgdorferi

Lyme disease is the most prevalent tick-borne infection in North America and Eurasia. It is caused by the bacterium Borrelia burgdorferi and is transmitted to humans via the bite of infected ticks. These spirochetes can cause both acute and chronic infection and inflammation of the skin, joints, heart, and central nervous system. The persistence of infection despite the presence of an active immune response is dependent upon antigenic variation of VlsE, a 35 kDa surface-exposed lipoprotein. A large number of different VlsE variants are present in the host simultaneously and are generated by recombination of the vlsE gene with adjacent vls silent cassettes. To try to identify factors important in vlsE recombination and immune evasion, we selected mutants in genes involved in DNA recombination and repair and screened them for infectivity and vlsE recombination. Mutants in genes encoding RuvA and RuvB (which act together to promote the exchange of strands between two different DNA molecules) had reduced infectivity and greatly diminished vlsE recombination. In immunodeficient mice, ruvA mutants retained full infectivity, and no vlsE recombination was detected. Our findings reinforce the importance of vlsE variation in immune evasion and persistent infection. Continued

HLA-DR alleles determine responsiveness to Borrelia burgdorferi antigens in a
mouse model of self-perpetuating arthritis.

Iliopoulou BP, Guerau-De-Arellano M, Huber BT.

Tufts University, Boston, Massachusetts.

OBJECTIVE: Arthritis is a prominent manifestation of Lyme disease, which is
caused by infection with Borrelia burgdorferi (Bb). Chronic Lyme arthritis
persisting even after antibiotic treatment is linked to HLA-DRB1*0401 (DR4) and
related alleles. In contrast, patients whose Lyme arthritis resolves within 3
months postinfection show an increased frequency of HLA-DRB1*1101 (DR11). The
aim of this study was to analyze the underlying mechanism by which HLA-DR
alleles confer genetic susceptibility or resistance to antibiotic-refractory
Lyme arthritis. Continued